2018 August FSM Practitioners Webinar – by Dr. Carolyn McMakin
Hi folks, we’re good? Yeah, hello this is been an incredibly busy month, we did the Denver seminar and then I had cataract surgery on August first and then most of you know the doctor George broke his hip on July first and had it replaced on July second and because we use new fracture protocol before the ambulance even came he had no bruising and took a total of, I think three Tylenol for hip replacement surgery which was pretty darn amazing. Anyway so he's doing great and then I have my second cataract surgery on the fifteenth which as it was five days ago. So I can see clearly now, there's a song like that right? If you're my age.
Anyway so there are many case reports this month, there's a great case series from Cleveland Clinic that they have on their website that I was going to show you and if we have time when we get through this I might actually do that. It's a PDF of their slide presentations of some of the complex cases they have been treating but what it made me think of was how it is that we learn FSM, how I teach it and by teaching you the language, how to think about it when to use it and I think all of us can do with a reminder, at least that's what this webinar is about this time, is a reminder of how we use what frequencies and I made some slides for a webinar for Functional Institute for functional medicine and went through the FSM history and I thought you might like some of the pictures. So I put them in the front, you know the history pretty much.
Frequency Specific therapies were developed in the early nineteen hundreds by early MDs and osteopaths in the US, the UK, Germany, those are the major ones and used by thousands of physicians from 1934 and we know medicine labeled them all as ineffective fakes drugs and surgery are going to be the tools of medicine.
This is what happened in about 1910 to 1934, during that twenty years they were trying to standardize medicine and nutrition, herbs, homeopathy and frequency therapies, electromagnetic therapies were all outlawed and any physician who use them would lose his license to practice. So the devices went into the backgrounds, the research was lost, you could find some of it in the rare book room at the Naturopathic College and the practitioners were persecuted, a number of them ended up in jail, they lost their licenses it was a thing.
Harry VanGelder bought a practice in 1946, went into the back room found this machine under the sheet and took the sheet off and found a device that was made in 1922 and that device came with a list of frequencies. The list was resurrected in 1995, George Douglas went down and worked with Harry 1983 brought the list back and stuck it in a drawer and when I started practice in 94 he bought a perception micro, two digital blue box, brought a precision micro device and we hauled the list out, he found the list and in 1994, 95 really we started using the frequencies with the blue box.
We really don't know how they were derived, we’re uncertain about the mechanisms of action but we've got a pretty good model for that now. What we do you know is the 1920 equipment was not microcurrent, frequency specific microcurrent is not about the device anyway, it's about the effect of the frequencies, so the frequencies were first taught in 97 and those of you that survived that era know that they weren't taught very well but the benefits, the effects and the effects of the frequencies are consistent teachable and reproducible research in animals and humans and clinical results have accumulated over the next twenty years. So this is the intro that I did for a webinar for Institute for Functional Medicine and their energy module which is really pretty fun.
So we know the frequencies work they produce predictable reproducible results but how do they work? What are they doing? The data gives us an idea about how the frequencies might work okay. We know that 40 Hz reduces information right, we’ve got the animal research with the mice and the ears and 62% reduction in LOX and the 30% reduction COX and is four-minute time-dependent response which is a feature that suggests the model we have, the cell signaling model we have is correct because it takes about four minutes to change what the cell does for a living. All of the inflammatory cytokines went down but then it makes you think where does information come from?
The immune system, where does it come from? The immune system responds with inflammation to either trauma or infection when tissue products from trauma or bacterial or viral cell fragments land on this cell membrane receptor. The receptor activates kinases inside the cell. The kinases activate transcription factors, is this appearing in your screen? The red and the black square? Are those there or are they just in mine? Okay. So the kinases activate transcription factors, the transcription factors change genetic expression, it changes what the genes do inside the cell okay and cause the cell to secrete inflammatory products.
So that's where inflammation comes from, it actually is produced by specific genes in the cells in any particular organ or tissue that are programmed to secrete inflammatory products in response to certain external factors that change the way the cell operates, what it thinks about, what it is told to do by these receptors okay, so that's a background.
So we know the frequencies reduce inflammation but how is that done? So 40 Hz reduces all inflammatory cytokines in medicine inflammatory cytokines are hard to change and when they change they change slowly over months but 40 Hz on A and 10 Hz on B reduce all of the inflammatory cytokines by ten to twenty times in ninety minutes but it only worked in patients who had spinal cord inflammation. This patient here at the control patient, she didn't have any reduction in pain, she didn't have any reduction in cytokines because her cytokines weren’t elevated to begin with. Forty and ten didn’t w work on her, it didn't reduce her inflammation because she didn't have any, and it didn't create a change in her spinal cord because there was nothing wrong with her spinal cord. Her pain was all peripheral even the substance P side on her is flat line. This is the placebo patients but this is the myofascial pain patient that didn't have anything wrong with her cord. So forty and ten didn't do anything to her, forty and ten only works in patients whose pain comes from inflammation in the spinal cord. Forty Hertz reduce inflammation ten is the spinal cord. How does that work?
If you haven't thought about that before I'm not surprised because it's a little… makes your brain hurt but that's what we're going to do for the next sixty minutes, so you know buckle up this will be fun, well in an interesting sort of way. Alright so cytokines are created by changes in cell signaling right, IL-1, IL-6 TNF-alpha, IFN Gamma CGRP, all of these cytokines have changed in that research, are all created by changes in kinase receptor configuration, kinases is transcription factors genetic expression. That's what changes, increases inflammation and our hypothesis is the only changes in cell signal could normalize them so quickly. We simply turn them off by changing the receptors and changing genetic expression.
Alright so the basic rule of science is you can’t throw out the data because it doesn't match your model right, you have to change the model so it can explain the data. In twenty years we've accumulated a fair amount of data that was a hand raise, so will get to the questions in a bit okay. So drugs or nutrients act like keys in a lock to change membrane receptors and intracellular function, right? We know how those work Advil, Ibuprofen lands on this receptor, modifies its configuration and changes prostaglandin secretion or pro-inflammatory cytokines or prostaglandin and it’s a prostaglandin inhibitor okay that's how Ibuprofen works, this way to change what the cell does for a living. Now the frequencies act like your key fob, your key remote opening lock with an electromagnetic signal. They appear to change, I don't have any research, genetic research to prove this but it’s the only thing that makes any sense and at least it is a testable model, if anybody knows anybody who has a genetics lab that would be interested in testing this out I’d love to see it proven. So the frequencies appear to change, membrane protein configuration and cell function electromagnetically not with a can a lock but like your key fob opening your car door, I couldn't remember when I…
Okay, so the model, our model, the model for how it works, our model says that frequencies change cell function by changing generic expression. So here's the thing if abnormal cell signaling is the cause of disease, pain, and illness and if frequency is change cell signaling in many different tissues what can the frequencies treat and how do you think about the frequencies in order to apply them to the situation?
That's what this webinar is about, is how to think about it okay. Genes are turned on by, let's say turned on by injury determine the rate of feeling right. So the frequencies change those genes and speed up healing. So we've got this post facelift treatment, Merissa Brennen this is her patient. So day one, two, five it's eighty minute treatment immediately Post-Op, daily for seven days and this is what this patient looks like at eleven days that's not possible, anybody that has ever seen a facelift patient two months after a facelift she might look like this but she's not going to look like that in eleven days.
So how is it that we're doing this? It's really fun to be able to do it, it’s predictable, reproducible, anybody that's listening right now, that's taken a course, seminar, read the book can do this but what are we doing? How does that work? Terrell Owens, open spiral fracture, torn interosseous ligaments, torn deltoid ligament. FSM for four weeks, he was healed and stable at four weeks and then a five and a half weeks we took apart the scar tissue and he played in Super Bowl six weeks after a career-ending injury and surgery. How did that happen? How did we do that? It's nice that we can do it and it's nice that anybody that's listening can do it, it's nice it's reproducible but how did we do it? We aren't using the frequencies to change genetic expression but the only thing that new injury treatments have in common is this four-hour window, right? You need to treat new entries within forty-eight hours, four to six hours after the time of the injury in order to get the optimal magical new injury response right, we call it the four-hour window. Thanks to Diana Cross we know that the only thing that happens in the first four to six hours that is unique is these early response genes are turned on in a new injury. They come on immediately and they are off at six hours, four to six hours. So we have to treat the patient in the first four to six hours in order to modify what these genes are doing. That's an observation, right? That's what science is about, it starts with observation. So we have this observation, you have to treat them in four to six hours, what is different between hour one and hour ten? The only thing that’s different between hour one and hour ten is these genes.
My model, our model says that frequencies change cell function by changing genetic expression but the new injury treatment focuses on 18/62 to stop the bleeding and reduce inflammation 40 Hz and repair tissue damage 124/294/321/9. These frequencies don't change the genes, they change the cellular process that turn these genes on. Look at the graph time zero when the genes get turned on what turns them on? Trauma, injury right, what is unique about trauma and injury? In general, there is bleeding, you damaged tissue, you break blood vessels and there's bleeding, the tissue is torn and broken and that stimulates information and that turns on this whole cascade.
We're not treating to change the genes but we are treating with the frequencies that turn off the cellular processes that turn the genes on, you have to treat the cause, what causes these new injury genes to go on, bleeding, inflammation, tissue damage. We use those frequencies to treat the cause and change genetic expression.
So the cell responds by changing genetics expression once the stimulus for the dysfunction is gone the drivers that create cell dysfunction go away and the cell can go back to normal function, as long as it has the resources that it needs. That takes us back to the whole stable state strategy, so the drivers that create cell dysfunction, have you ever wondered why the frequencies for toxicity reduced pain? Ever wondered that? I wonder that, have you ever wondered why the frequencies to remove mold or pus and pus and capsulated, why does that change pain? Are we killing mold? No, I don’t think so, are we killing bacteria? I don't think so, the frequencies are too low they’re not complex enough.
What do the frequencies do? If the frequencies are changing cell signaling and if toxicity or mold or pus and pus capsulated or strep or stuff or pathologic virus, if those frequencies are modifying the receptors on cells and those receptors are changing transcription factors and kinases inside the cell to create inflammation, or reduce secretions or do whatever they're doing and we change the drivers that create cell dysfunction once we change the effect of mold or the effect of infection on this receptor the cell can go back to normal function as long as it has the resources it needs to do that.
So there has to be a stable state to support it, this kind of all hangs together when you look at it as a big picture, we'll do some examples in a little bit. So torn or broken in the tendon right, do ever think about why torn or broken in the tendon works for tendonitis? Because it's not inflammatory, tendonitis is not actually inflammatory, it's a tendinopathy. It happens when the tendon is injured and can't be repaired within about twenty-four hours to forty-eight hours, the cell begins to express the genes to secrete inflammation. 40/ 191 reduce inflammation and the tendon doesn't work, never did. I used it for a year, it was my tendon, and it didn’t work, why? Because 40 was not the problem, I could reduce the inflammation but it only lasted twenty-thirty minutes, the engine tendon is inflamed and swollen but torn and broken and the tendon fixes why. The tendon cell is secreting inflammatory products, it fixes why the tendon cell has turned on the genes that produce inflammatory products.
Why it's time-dependent I think it just takes that long to fix the tendon. It takes about an hour to two hours, those of you that have treated tendons, ligaments and connective tissue know that it's time-dependent but when you think about why it works, it treats why, it treats the driver that's driving the cell to be dysfunctional. Tendinitis are not supposed to express the genes for inflammatory products unless they're injured. When you remove why the tendinitis is secreting the inflammatory products, why the tendinitis is expressing the genes to create information. If you fix the driver you change what the cell does, it’s kind of an interesting way to look at it really.
Alright so we're going to look at examples of this as we see it in the brain mostly with the sensory and motor cortex 92 and the midbrain 89, these are all from core seminar okay. So frequencies for the brain and we're just going to look at really 92 and 89 in various case reports because it's just the last couple of years has been fascinating. So Thalamic pain syndrome from strokes right? We get rid of thalamic pain, thalamic pain syndrome, and central pain 40/89 you run that neck to feet. We know that 40 Hz reduces inflammation, I don’t have any information that tells me that thalamic pain syndrome is caused by inflammation in the thalamus although it could be because it's a response to a stroke right? So once you have a stroke the cells are ischemic, they die, they’re dysfunctional, there's information. So reducing inflammation of the thalamus would reasonably reduce pain but it also appears to reduce the activity of the target tissue.
So central sensitization, when you treat somebody that has 40/10 pain fibromyalgia from spine trauma your reducing inflammation in the spinal cord, we have data that says that. Once their pain gets to a zero they look confused, right? They’ve got a funny look on their face because the thalamus is still sensitizing or amplifying pain, either there's no brain injury, they haven't had a stroke but the thalamus is hyperactive. So 40 Hz, in that case, appears to reduce the activity of the target tissue, in this case, the thalamus but it also works in Phantom limb pain, what's up with that? 40/89 is how you get rid of phantom limb pain right? You treat neck to feet, it doesn't matter which limb is missing you can treat neck to feet, if it's the arm or any part of the leg that's been amputated, 40/89 will take down phantom limb pain, it takes about thirty to sixty minutes but does the patient have a brain injury? No, so as the thalamus inflamed? I'm not sure, I don't think so. So we could say that 40/89 reduces the activity of the thalamus in response to missing input from the missing limb right?
That's one way to do it and then treat local pain at the stump by treating reducing inflammation in the nerves that make sense and then you treat bruising and inflammation and scarring in the soft tissue, the fascia and the muscle belly and the periosteum and the amputated limb. So 40 reduces the inflammation for sure but it also reduces the activity of… I should have put in the size from Roger Billicas heart rate variability study. 40/562 quieting the activity of the sympathetic, reduced sympathetic response and 40/709 reduced the activity of the parasympathetic and he could manipulate that well. Up or down just by running 40 or 81 interesting isn’t it and then 81 increases secretions. This was what caught constructive interference, it supports the function of the secretions in the tissue for which it's listed. So this is the ovarian study right, so salivary estrogen at two thirty was 1.7, at five o'clock it was like 1.4, treated pituitary increase secretions and pituitary didn't change salivary estrogen and between five thirty and six we ran increase secretions in the ovary and that increased salivary estrogen from 1.4 to 37.1.
So that is clearly not a placebo effect, something is real that's happening when you run 81, so somehow 81 Hz changes the secretory activity, affects cell signaling in such a way as to turn on the genes. So it changes that receptor to turn on the genes that cause an increase in secretions that are specific to the organ that's targeted. I want to say that again because I don't have a picture of the genes here, but 81 Hz increases secretions I think by changing the receptors on the cell that increase by changing the receptors on the cell that change the genes or turn on the genes that are specific for the secretions from that tissue. It was only 81 Hz on A and 7 Hz the ovary on B that increased salivary estrogen.
Alright so let's look at where we use this, this is a fibromyalgia patient, he had rigidity in the peripheral muscles from a spinal cord injury that happened when he was five years old, and his muscle tone was incredibly tight everywhere from the neck down, his legs were like bricks, now clearly he works out, he exercises a lot but 40 Hz on channel A and 10 Hz on channel B took down his body pain but made the Spasticity worse. 81 Hz on channel A increase secretions in the spinal cord, relax the tone in his muscles, starting from the bottom up so the legs go first, lower leg goes first then the quadriceps, then the trunk muscles, then arms and then the hamstrings go last. So this is a really consistent pattern, we've been doing this for about ten, almost ten years now. What's it doing though? Think about it, what's it doing? The only way this works, is if somehow 81 Hz changes a receptor in the spinal cord, that receptor somehow increases or changes the transcription factors in the genes and causes an increase in secretion of dopamine down the spinal cord to increase descending inhibition, I think it's dopamine, could be something else.
I'd love to have some data on it but it increases secretions in the spinal that facilitate descending inhibition of spasticity. Spasticity in the periphery, this increase in tone that happens in patients with myelopathies, spinal cord or brain injuries the spasticity goes what? It is there because of loss of descending inhibition, there's a constant dial tone, it comes down from your brain, this is down spasm, when that dial tone gets interrupted by something you have spasticity, it works and cerebral palsy, it works in myelopathies and it worked in this patient.
So 81/10 reduces spasticity specifically that specificity that is caused by a loss and descending inhibition from the brain down the cord to the body, you still with me? I told you this makes your brain hurt, this makes mine hurt. Alright, now 40/92. So the sensory and motor cortex I’d put a picture of the brain here but we just did that right? Sensory-motor cortex strip on the cortex, so 90 is the cortex 92 is the sensory and motor strip in the cortex specifically.
If you ever have a patient that is super ticklish like you go to work on their abdomen or their armpits and they just bust up giggling and they can't sit still and they’re super ticklish run 40/92, just switch the frequencies to 40/92 and inside of thirty seconds to a minute ticklishness is gone and you can do whatever you want. I’ve done it enough times that I can say that with some certainty.
However when you have children who have difficulty with sensory integration right? They're super sensitive to all sensory input taste, sound, touch, it does seem to work in autistic children but this is that subgroup, we call then sensory integration patients 40/92 reduces that and then if you treat ninety-two for concussion and reduce inflammation see what happens.
Try to run yourself sometime, run 40/92, run 81/92 and see what you notice, what happens to the colors in the room? What happens to your sensitivity to sound? What happens to how much sound bothers you? Okay, when patients have inflammation body wide from a viral infection or GI problems or whatever, when patients have inflammation you notice that you are light sensitive and sound sensitive when you have a migraine light and sound sensitive, where does that come from? I'm going to suggest that it's this, 40/92 because you can change it with the frequency. 81 on channel A and 92 on channel B, the sensory and motor cortex.
This young lady had a stroke, if you were here for the May I think it was webinar this is a young lady we treated in Taiwan, she was thirty-eight when she was thirty-five, she had a stroke because she had sleep apnea and her hand was clenched, spastic, flexed her shoulder was abducted, flexed, spastic and immobile. We did increase secretions in the sensory and motor cortex and look what happened. The spasticity in the arm and the hand relaxed, pretty soon she could mobilize her own hand. We ran increased secretions and Kathleen Kasper the physical therapist could mobilize her shoulder and it relaxed and I figured well we've gotten everything we can out of 81/92 so I switched to 49/92 and then they did this and treating the sensory and motor cortex to remove trauma increase secretions and restore vitality this was permanent. She hadn't moved her shoulder in three years because these muscles here were spastic.
The second treatment, this is what was lasting from the first treatment and the second treatment did this. That's what happens when you increase secretions in the sensory-motor cortex but what are we doing? Why does it work? What is this spasticity caused by? Spasticity is caused by the damage to the sensory and motor cortex that from the stroke that stop the secretions from the sensory and motor cortex, motor cortex particularly, it stops the secretions from the inner cortex it tells us area not to spasm, okay.
So when you increase the secretions you get rid of the spasticity, we have much better luck getting rid of spasticity than we spastic paralysis that we do getting rid of flaccid paralysis. It’s a different frequency or something because flaccid paralysis I haven't had a lot of luck within the spinal cord or the brain but spastic paralysis increasing secretions from the sensory and motor cortex appears to connect it to the periphery. Now she has to have a stable state right, that includes enough protein and amino acids to make dopamine and protein and amino acids to make acetylcholine right dopamine in particular. 81/92 when you treat and RSD or CRPS patient, so if you've seen the video of me treating Adam for say CRPS in two thousand whatever that was fifteen, fourteen.
The way you get motion back in RSD CRPS patients and other denervation patients is to reconnect them by increasing secretions in the sensory and motor cortex, what are we doing? Somehow we have to be affecting receptors on the cell that change the transcription factors, that change the genetic expression and cause this cell to starts secreting the peptides associated with motor function. With me still? Okay, hang in there. So this is from Regina Thompson, she is a physical therapist at Cleveland Clinic and we've had an ongoing discussion of…if I finish early enough I'm not going to finish early enough I’ll show you the presentation that is on the Cleveland Clinic website that they've done with spasticity and treating different neurologic conditions and their pediatric patients.
So this is the pediatric rehab hospital and these folks see some very difficult patients. So we're talking about reducing spasticity by increasing secretions in the spinal cord. 81/10 is the… this from an email I got from her today, 81/10 is especially good but nothing beats 40 reducing inflammation, pain reactions, spasticity than increasing secretions and vitality in the spinal cord but look at this it's five minutes each, it doesn't take long. When what you're doing is changing cells signaling, you change a signal it's like opening your car door with your key fob. It takes five minutes to do this, reduces spasticity every time, we use a long diversion of that for tougher cases. So the more difficult and refractory the damage to the cell and the dysfunction and the receptor the more difficult it is the longer it takes to what? To change this, to change the genes, to wake up the genes, I would love to be inside a cell, I would love to know what we're doing. She also uses 81 increase secretions, 988 is from the basal ganglia, is from the advanced, it’s for the basal ganglia. So she also uses increasing secretions in the basal ganglia and the cerebellum to improve coordination and motor planning. So this has been very successful on a child with [inaudible 38:22] is a dysfunction in the brain basically underdeveloped basal ganglia and she's going to find that because the protocol has been so effective, however, this is from Ben Katholi.
Dystonia patients are hypertonic right? They have too much drive from the basal ganglia, so it makes them spastic, there's too much, that's what dystonia is, it's too much. So increasing secretions in the basal ganglia makes the tone worse but quieting the basal ganglia and treating for trauma in the base of ganglia is pretty cool.
So five years ago these guys took the course seminar and they learned the language and then they have a place to use it. I don't know that we think about the fact that we're probably changing cell signaling, most of us in medicine are pragmatic. It's is it going to work? What does it do? But it makes you think right? Do you want to quiet the activity of or increase secretions? You'll know in about three to five minutes whether or not it's going to work but it lets you think about what it is that is causing the patient's symptoms. Increased secretions in the basal ganglia made this Parkinson's patients go from spastic and trembling to not spastic. It basically effectively reduced his Parkinson's symptoms by about 80%, it's not going to hold because there's no stable state, there's not enough cells in the basal ganglia there to repair and he's way over medicated but just the fact that we could change the Parkinson's symptoms by treating the basal ganglia for toxicity right, to help change the signaling, help change the signaling that has been damaged or affected by toxicity and fibrosis inflammation, chronic inflammation, scarring.
Whatever you think is impeding the function of the cell that is driving the change or the dysfunction in this receptor. So in Parkinson's, we know it's associated with toxicity, sometimes viruses but toxicity. So you want to treat that to remove the effect of the toxins but you also want to drive secretions in the basal ganglia. So our model says that frequencies change cell function by changing genetic expression, so if abnormal cells signaling is the cause of disease, pain, and illness and if the frequencies change cells signaling in many different tissues what can the frequencies treat?
Kind of makes you think that we can treat just about everything which I think is true but then how do you think about them? This, it's a good thought experiment, what you know that patients that have spasticity if you run 40/10 it will make the Spasticity worse. If they have body pain and you run 81/10 and what you're increasing secretions of is substance P you're going to make the pain worse, but 40/10 will take the pain down and 81/10 will take the spasticity down and I have had patients where I had to run both at the same time, that's a thing that knocks me out. You can do both at the same time, I've run 40/10 on a patient and at the same time run 81/10 in the same patient and the pain went down and the spasticity went down at the same time, it's kind of mind-boggling right?
So you change the pattern by changing cells signaling, treat the cause by changing cell signaling and by supporting the stable state. So if you are trying to fix somebody with atrial fibrillation you have to increase secretions in the vagus nerve right? Is there any point in doing that if the patient doesn't have enough choline? Acetylcholine is what the vagus nerve uses as a non-transmitter right? If the patient doesn't have enough choline will you be able to sustain that treatment of atrial fib? No, so supporting the stable state yes you can make a change.
Toxicity in Parkinson's support the stable state, he's not around the toxins anymore but you have to help repair the mitochondria with lipoic acid and co-Q ten, all of the sub stable state stuff that we know to do. It’s totally fascinating, well we're just about at the end, this is so much fun I love these things. Today was really fun, I had a great time, hope you did too, hope it didn't make your brain hurt.
We've got three seminars left this year in the US. We're going to be in London September 13, 14, 15 and 16 and at the Osteopathic College it's down by basically down by London Bridge it's just a lovely area of town. Then we'll be in Jacksonville on October 25th to 28th, Minneapolis November, and San Francisco in December and I encourage you to come and take another course seminar, it's different every time I teach it and these concepts of what causes, how are using frequencies and what they're actually doing.
I love being pragmatic and I just like fixing stuff but every now and then the mind wanders to what it exactly is it that we're doing.
And just a reminder there's a new FAQ brochure, it’s updated information, it's easier to read, it's a lot prettier and I cleaned up some of the text, so that's lovely. We print them off, Shelton Turnbull does the printing for us and we sell them just above our cost, I think for a while there we were losing money on them. So you can buy them from us or we will actually send you the PDF and you can have them printed yourself, I'm not proud I just… these are really useful for patients so that they know what you're doing and what services you have to offer with FSM.
I'm going to encourage you also to share the Resonance Effect with your patients and colleagues, it is proven to be really readable and patients like it. So it will give your patients a better idea about what you do and why you do it and what's possible and how the effect of frequencies and Resonance can be used to help them and what you're doing with it.
So that's pretty exciting. I know what I forgot to put in here, was the practitioner list subscription, website practitioner list subscription. Kevin will be sending out a notice probably about once a week so that it's only eighty-nine dollars a year. Patients are looking for you to come to you to get treated and we had to start charging for it so that there was a way of renewing your subscription so that we could keep the list accurate and up to date. Eighty-nine dollars a year if you get just one new patient off of it, it will be worthwhile and it's good inexpensive advertising. So I recommend that you sign up for the practitioner, website practitioner list if you haven't already done that and then 2019
The Advanced and Symposium is coming up, the course seminars March seminar is 14, 15, 16, 17 at the Crowne Plaza San Marcos, it's in Chandler actually just outside of Phoenix. 18th and 19th Kim Peters is doing the sports core right after the core seminar.
The 20th is the instructor training, so the FSM practicum instructors so you can help us out at core seminars and ultimately so that you can do the practicum instruction for students who have taken the course by textbook or on DVD. Our instructors are really crucial part of the spread and sustainability of FSM. When we had the core seminar in Denver with forty-two students I had, we had four instructors Sue Spizer, oh come on and see faces so I can't remember names but never mind Miller Miller and the nurse practitioner yeah Kevin's fried too, sorry I can see your face anyway. It'll come to me as soon as I stop talking anyway.
The 21st and 22nd is the Advanced Course. Its new frequencies and treatment strategies, we've got your favorite faculty will be back I hope. Expert workshops that are ninety minute workshops and then I do the basic advanced stuff in the mornings and then 23rd and 24th is the FSM Symposium and this year the speaker list is just Stellar, Jim Oschman will be back, Gerald Pollack who wrote Cells Gels in the Engines of Life, he also wrote The Fourth Phase of Water, Gerald Pollack is going to be there, he'll do a ninety minute workshop. Juliana Mortensen is a medical physician and an MD PHD who has actually patented frequencies to kill viruses and I want to hear that story. She does a great lecture on the physics of how frequencies could be doing this but I'm going to see if she'll grace us with some information about what her virus frequencies did and how she used them. Jim Turner is the attorney that helped us helped us make it through our FDA audit and Diana Cross will be here from Australia. I don't know what she'll talk about yet but every time she comes she makes the sun come up, she opens up a door that then feeds what we do for the next three to five years and then there are practitioner case reports that are pretty exciting.
Remember that there's a thousand dollar prize for any practitioner who publishes a case report and we have two of those to give out this year. Regina Thompson has a paper published and there's somebody else with a paper in the pipeline. So if that is published by March there will be two thousand dollars checks to give out, be sure and consider publishing a case report and any sort of peer-reviewed journal or collect a case report, that's how we are going to make ourselves sustainable, that's how we earn credibility for your practice and for FSM.
So here's what you’re doing, we're changing medicine, we're doing it one patient at a time, one stroke patient, one fibromyalgia patient, one dystonia patient, one spasticity patient and each one of those patients that are treated successfully changes their lives and every practitioner, everybody listening and everybody that contributed to the results in this webinar one practitioner you, just you one practitioner can change lives in ways that will constantly surprise you, blows me away all the time and changing even one patient's life can change the world. So thank you for joining us enjoy what you’re doing and thank you for making the world a better place and will see you next month.