Episode Ninety-One (Carol McMakin’s conflicted copy 2023-06-15).mp4: Audio automatically transcribed by Sonix
Episode Ninety-One (Carol McMakin’s conflicted copy 2023-06-15).mp4: this mp4 audio file was automatically transcribed by Sonix with the best speech-to-text algorithms. This transcript may contain errors.
Dr. Carol:
Hi.
Kim Pittis:
Hi, I missed you.
Dr. Carol:
I missed you, too. I managed. I had an opening theme, and since I had, like, literally no idea what I was going to do, the theme for the day was flexibility.
Kim Pittis:
Perfect.
Dr. Carol:
And improvization.
Kim Pittis:
Yes.
Dr. Carol:
One of the practitioners gave us the closing quote, and I don’t actually remember what it was, but it was really good.
Kim Pittis:
I love it’s like a little village that we have. So everybody like stepped up and participated.
Dr. Carol:
And we got to answer questions, and it was really fun. Did you have fun in Canada?
Kim Pittis:
It was very busy as all the trips up to Canada are. We were on the West Coast for a good portion and then, we were on the East Coast for the other portion.
Dr. Carol:
Oh my.
Dr. Carol:
And just got back yesterday and my cherry tree is exploding.
Dr. Carol:
With cherries or with blossoms?
Kim Pittis:
With cherries.
Dr. Carol:
Oh, my gosh.
Kim Pittis:
I wish I could send them to you because they are like the size of plums, and they’re deep red and they’re so sweet. And it’s the biggest harvest we’ve ever had.
Dr. Carol:
No way. That’s amazing!
Kim Pittis:
It’s just the little things when you come home and then there’s a cherry tree that’s literally bending with cherries.
Dr. Carol:
Oh, that is outstanding!
Kim Pittis:
I missed everybody. I missed doing this.
Kim Pittis:
And no, leif I didn’t come visit you in North van, Calgary, was as close as I got, but…
Dr. Carol:
That’s okay.
Kim Pittis:
I’ll let you know the next time I’m up there.
Dr. Carol:
And I got an email from somebody that listened to the podcast. I’m not sure when it was because I don’t remember saying what she said I said She said, I said, don’t be so Canadian and I would never say that to you. I can imagine saying you are so Canadian.
Kim Pittis:
I think I remember that. But it was like when I was treating you and you thought my hands were polite or something like that. And I think was that the reference?
Dr. Carol:
It was on the podcast.
Kim Pittis:
Oh.
Dr. Carol:
But I love that you’re Canadian.
Kim Pittis:
I’m officially dual. I have my American citizenship now, so I’m like, I get.
Dr. Carol:
The passports.
Kim Pittis:
what did you talk about? I want to talk about the possibility.
Dr. Carol:
No, you’re too quiet. I don’t actually remember. It was all Q&A. So it was random stuff.
Kim Pittis:
Yes.
Dr. Carol:
And I don’t actually remember, but.
Dr. Carol:
We answered a lot of questions, which it was like a masterclass without.
Kim Pittis:
Yeah. Fantastic.
Dr. Carol:
But I have to tell you, I have patient stories from this week.
Kim Pittis:
Okay, I need to.
Dr. Carol:
So there is a condition that is genetic. It’s called SPG7 and a patient came from out of state. Can’t remember where she came from, but it’s the corticospinal tract. So the motor pathway and I think of the corticospinal tract in the spinal cord. When you look up in your phone and yeah, the corticospinal tract goes up your spinal cord, but then it goes Medulla to PONS to sensory motor cortex. And the symptoms are motor weakness, spasticity, foot drop. Her tongue was numb. Her voice was fuzzy, and she didn’t have a strong bladder contraction. So, when she went to urinate, it just dribbled out. She couldn’t activate the muscles in her bladder.
Dr. Carol:
So you look it up and you go, okay. We have a frequency to repair DNA. And the good news was that her genetics were heterozygous. She had one copy of the gene that was present and one that was absent. And she was fine until she was 35. And now she’s 40 something. So she’s been like this for 5-6 years Anyway, so while we had one copy of the gene, she was fine until she was 35. So what happened when she was 35? A bunch of stress.
Dr. Carol:
So, we ran concussion in Vagus. And then if you have one working copy, you can drive it to produce Gaba and acetylcholine. So, acetylcholine is for the PONS and the mouth and the upper nerves and then Gaba is to reduce spasticity. Anyway, I looked it up and went, Can’t hurt. Might help. Hooked her up from neck to feet with sensory motor cortex, pons, cerebellum, medulla, spinal cord and nerve. All with increased secretions. The first day I didn’t do the nerve and the foot drop didn’t improve. Second day, I did the nerve. She urinated forcefully. Her tongue was never numb. The tremor in her hand went away and her gait settled down. Foot drop got better. And I took her into the gym and we ran 81/84. Increase secretions in the cerebellum to coordinate everything. Yeah. Nice that you’re not spastic anymore, but your brain has no idea how to operate these muscles to make them work.
Dr. Carol:
So she got so stoned when I did increase secretions in the cerebellum. It was hard for her to walk, not because she wasn’t coordinated, but because. So we’re walking down the hallway and there’s that sign in the clinic, “Be realistic. Expect a miracle, but be patient. The impossible takes longer than the difficult.” What we did in that 60 minutes was impossible.
Dr. Carol:
She started sleeping eight hours a night. Instead of waking up with spasticity every 2 or 3. It was, okay, fine.
Kim Pittis:
And that was one treatment?
Dr. Carol:
She was there for 4 or 5 days.
Kevin:
Okay.
Dr. Carol:
Five days. And we had a weekend. So the last treatment was on a Friday and every day got better. It held for longer and then she took a CustomCare home with her on Monday. And because her legs are working better, but her trunk is still having trouble and she’s not moving her arms, it took me until Monday to tell her I programed a combo that she can run at night and then programed each frequency separately. I said put 81/84 increase secretions in the cerebellum on your neck and put wraps on your ankles and crawl. You won’t fall and you have to get your arms and your legs coordinated.
Kim Pittis:
Yeah.
Dr. Carol:
That was last week. This week, there is a girl that came in with complex regional pain syndrome. So basically neuropathic pain in the superior laryngeal nerve which operates the vocal cords. So, she hasn’t been able to speak without intense nerve pain for a year and a half. And it was mold, then Covid, then Lyme, over a 3- or 4-year period. So it’s okay.
Dr. Carol:
CRPS means the vagus nerve is disconnected. So we ran mold in the Vagus. Virus in the Vagus. Lyme in the heart. Took her heart rate from 108 to 81. And then, Lyme and the Vagus, increased secretions in the Vagus and at the end of 60 minutes, she was able to talk without pain for the first time in a year and a half. So that was my week so far. And it’s Wednesday.
Kim Pittis:
Okay. I just had a bunch of athletes that PR’ed everything that they’ve ever touched, so nothing compared to these stories.
Dr. Carol:
I asked her, what’s your official diagnosis? Because she’s going to submit to get the CustomCare paid for and she said they diagnosed me with CRPS in the superior laryngeal nerve. And I said superior laryngeal nerve is the Vagus. And she went, Oh, the ENT didn’t know that. And I went, Oh, okay. So I showed them the vagus nerve webinar.
Kim Pittis:
Yeah.
Dr. Carol:
And we went through, she and her mom both had central sensitization from a very difficult childhood pregnancy, childhood. And it’s okay. There you go. Isn’t it cool?
Kim Pittis:
It’s funny and it’s not because Vagus was something I wanted to touch on a little bit because I have been doing like supine cervical with my athletes. They all have disastrous anterior necks. I don’t know why people have such issues treating the anterior C-spine or they think it’s not as important as treating the posterior C-spine. I don’t understand.
Dr. Carol:
They have to put together.
Kim Pittis:
But every time I work on somebody’s necks, people are just like, Oh, nobody’s really gotten in there ever. I’m like, okay, Why? How did you get through so many years of being a professional athlete without people seeing that this is very important structure? I don’t know.
Dr. Carol:
Without FSM, if all you have is being able to mash on it, you don’t want to mash on the carotid, the jugular, the Vagus, a little knot.
Kim Pittis:
True.
Dr. Carol:
On it.
Kim Pittis:
True. I don’t know. It does. And, yes, FSM makes it so much easier to just melt everything and to mobilize. And it’s hard for a patient to have people touch the front of their neck. It can set off a whole cascade of anxiety. And when they’re stoned, it makes things a lot easier to just move your trachea over and get into that longest coli or wherever you need to go. But I was working on a patient who nothing was letting go and part of my quote that I’m going to tell you now instead of after because it’s going to tie in together. The quote of the day today is “you cannot shake hands with a clenched fist.”
Dr. Carol:
Oh, Absolutely.
Kim Pittis:
So multifactorial quote here. But I had thought of that quote while I was working on this neck. And it’s no, listen to your own teaching advice. It doesn’t mean press harder or mobilize stronger. You’re missing it. And what let it go was 94/109. Trauma with the vagus nerve.
Dr. Carol:
Wow. Okay.
Kim Pittis:
And how it jumped out at me, I have no idea. But I got a little bit with scarring in the Vagus. I knew the Vagus was traumatized from the motor vehicle accident, from the fall off the horse, from the hit There were multiple things.
Dr. Carol:
Yeah.
Kim Pittis:
But it’s that voice that tells you you’re on the right B channel. But think about it differently. And we use 94. I’ve been really going back to those basics a lot. .And yet that vagus nerve, like what happened before the Vagus got scarred and I think this works for lots of tissues. If you’re getting some results with 13 so scarring in a tissue, you have to think it didn’t get scarred from outer space. What happened before it got scarred? There are some sort of trauma there.
Dr. Carol:
And in two patients this week, the problem was vagal.
Kim Pittis:
Yeah.
Dr. Carol:
But the place where the Vagus was stuck wasn’t here behind the ear. It was here where it crossed under and both of them had root canals. And so the place where the Vagus was stuck was the lymph node. So, the lymphatics that follow the Vagus down the neck, they ended up running an infection and scarring in the lymph node. And then, we could get the Vagus unstuck. So what is the Vagus stuck too? And it requires that you think in three dimensions and almost four dimensions because you have to add time. So, it’s not just anatomical three dimensions. It’s also time.
Kim Pittis:
Yes.
Dr. Carol:
What happened? Nothing. And then the key was which order and this particular patient had a bizarre root canal story and 3D cone beam that showed upper cavitations. So it’s one of the few people I know that started with Neil Nathan, Mary Ellen Chalmers, Dietrich Klinghardt, and then got to me.
Kim Pittis:
Wow.
Dr. Carol:
Yeah.
Kim Pittis:
So it cast right there.
Dr. Carol:
Yeah. And that was the problem. The Vagus got stuck before it entered the vocal cord, and every time she turned her head or swallowed because of the adhesions, she created a nerve traction injury in the Vagus and it eventually disconnected. But it had all these precursors. So, you’re exactly right. It’s like the frequencies let you think in four dimensions. Three physical dimensions. But also time. The auto-accident. The hockey stick. The horse.
Kim Pittis:
Yeah. Multifactorial. And then I had somebody ask a question. Why don’t you just start with trauma to begin with? And it’s. Yeah, I get that, but it’s not always indicated. And for me, I’m always thinking, how am I going to get this patient out of pain? So, I’m always thinking about calming the nervous system down first because it doesn’t matter where you think you need to go to, and you have those moments where you just start seeing numbers flash before your eyes because you know you have to hit all these things. But I always try to like take a breath and slow down and think, but I need to get this patient out of pain first and then, I can go back and make it permanent and make all those changes that I need to. So, we know that nothing is going to happen if our nervous system perceives a nerve is going to traction or terror. When there are structures around that nerve, fascia, muscle, adipose that are adhered and jumbled up, you have to address that first, regardless if trauma is what caused it, make a dent in it. I always use to think in my head, make a dent, get the smush, get them sleepy, get some movement going, and then you can go back and then, okay, we’re going to need to do this because . right there anyways.
Dr. Carol:
The body is not going to let you do anything as long as whatever you’re touching is painful. The muscles are splint up. They’ll bounce you off. For me, the anterior cervicals, especially the lower cervicals, it’s always 124/710.
Kim Pittis:
Right.
Dr. Carol:
In previous years it would have been inflammation in the disc annulus and then, two seminars in a row, the instructor said, I’ve been using 124 torn and broken in the disc annulus and that works better than 40. So during the practicum, we tried it and they were right. So, I’ll set one machine up at the anterior cervicals are really tight. I’ll set one machine up with 124/710 and I have single frequency combinations programed into a CustomCare. Find that one. Let that run for 60 minutes while I’m working on these facets and the muscles like what’s making the anterior cervicals tight.
Kim Pittis:
And that splinting mechanism that goes in the body regardless if it’s a disc, a nerve, degeneration and cartilage, like our musculoskeletal system is so smart and works at this amazingly subconscious level of always trying to think they have our best. It’s like a helicopter mom, right? We think we have our best interests for our kids and we’re going in trying to pad the room for them and it’s okay, stop. So that’s how I see sometimes all the splinting is unnecessary splinting that’s going on. And yes, treating like 13/77 scarring the connective tissue, you’re going to get some smush. Yes, put your little award behind you.
Dr. Carol:
I keep seeing your award behind you. And it’s. Wait, this is out of sight. i want to put it right? It’s the only time we look at it.
Dr. Carol:
Unnecessary splinting. The cerebellum thinks it’s necessary or it wouldn’t be there.
Kim Pittis:
For sure. And a lot of times it can work. Like with the torn labrum and the shoulder, that splinting is what takes the pain away. Otherwise, that labrum is going to continue to tear. It’s going to continue to get hurt. Again, going back to torn and broken in the annulus. So. 124/710, it is a magical frequency combination. It can be a religious experience when that’s indicated. And let’s face it, if we image the majority of people over the age of 35, we’re going to see some tearing in the annulus. So I think that’s a great one to start with because most of us have that at some level.
Dr. Carol:
Yeah. So I had a patient last week or the week before. She has lumbar stenosis, and she has a bunch of disc bulges and leg pain. And she said the side of my leg hurts. And she said, what about the herniated discs? And I looked at the MRI and yes, you have bulging discs, but bend forward. Does that make it better or worse? That makes it better. Okay, then it’s not the disc. What about the herniated disc? The orthopedic surgeon says it’s the disc and the stenosis is coming from the joints in the back. And that pain you have down the side of your leg, if you look at this pattern on the wall, it’s coming from the joints in the back. So the differential diagnosis, is it what’s the pain generator? Is it muscle, disc, nerve, facet joint? And then why is it hurting? Is it because you were a football player or because when did it get hurt? Is it still hurting? What can you do besides FSM? It’s really fun being us
Kim Pittis:
What were those tests when you’re trying to figure out what you wanted to do with your life? Not aptitude tests,
Dr. Carol:
Aptitude tests.
Kim Pittis:
And it would help you figure out what profession to go into, 100% of the time. Every time I do it, even to this day, detective shows up as like my and I’m like, Yeah, that’s what I’m doing right now. Every day I’m a detective.
Dr. Carol:
Yeah.
Kim Pittis:
I guess I’ve always had that question of why is this like that. And I think if you can really just start as a practitioner with that question, why is that patient presenting with this pattern? And you had said it a couple of podcasts ago about pattern recognition. You’re really good at pattern recognition and yes, doing something for as long as you have, you get that. But for new practitioners, I would say if you’re getting frustrated and we all do with what we are, if you can boil it down to that pattern recognition, asking the question why is it like that? And even patients that are listening, ask your body, ask yourself like, how did it get like this? What else was happening so that you can paint a better picture for the practitioner that’s treating you? Because there’s always that sub-story that’s going on that we tend to forget about because we’re not used to putting all these things together, I think.
Dr. Carol:
And then the other helpful question is what makes it better? What makes it worse?
Kim Pittis:
Yes.
Dr. Carol:
And that’s the key.
Kim Pittis:
Yeah.
Dr. Carol:
Because it’s constant when all day. No, it’s better when I get up in the morning. Okay. What makes it worse? Like hip pain as soon as I walk on it. Okay, let’s do an x-ray of your hip and there’s no hip joint left. So then I probably had, I don’t know, the almost double digits of patients who came in and said, I want to avoid a hip replacement. And you look at the x-rays and go, No. I can fix it so you can sleep at night. But these x-rays say that you’re about 3 to 5 years past the point where we could avoid it. I can’t put bone back or cartilage back that’s not there.
Kim Pittis:
Yeah.
Dr. Carol:
However, you hunt around, talk to your GP, your friends that are nurses, find interview for orthopedic surgeons who do only hips and pick the guy that you like. That will talk to you. That will communicate with that you trust.
Kim Pittis:
Yeah.
Dr. Carol:
So you’re comfortable going into it. And I have a CustomCare that you can buy. So, your recovery is six weeks instead of three months.
Kim Pittis:
Yeah.
Dr. Carol:
It’s easy. Not so much.
Kim Pittis:
Yeah.
Dr. Carol:
But hips, yeah.
Speaker2:
So funny that you mentioned hips. It’s one on my list today to talk about because somebody had who had taken the course had asked me about treating people with tight hip flexors. Yeah, my initial reaction is never a good one because I’m like, we need to get away from just hammering on people’s hip flexors because they’re never tight for no reason at all.
Dr. Carol:
It’s never the psoas. It’s always the ureter.
Kim Pittis:
Oh, there’s that. I will say biomechanically, we are living in an era, in an environment, where people have chronically shortened hip flexors, and we also chronically short legs, our anterior chain is chronically shortened because we’re sitting like we ever used to, but that doesn’t mean someone’s hip flexors are tight.
Dr. Carol:
Yeah, exactly. They’re always short. That’s a good point.
Kim Pittis:
And that’s what we talk about or you talk about the story all the time with I believe it was a marathon runner that came to see you and said that she had a weak glute. And you’re like, you run 30 miles a week. Like your glutes aren’t weak. Portions of it could be inhibiting.
Dr. Carol:
Glute is not weak. Your right glute is not going to be weak for no reason.
Kim Pittis:
So there’s that. And I’ll talk about the whole thing in a minute. But let’s get to a couple questions because I see they’re starting to pile up and I want to.
Dr. Carol:
Just pull them up.
Kim Pittis:
Okay. I think Leif had the first question about something about fat for diabetes.
Dr. Carol:
Hi, Leif.
Kim Pittis:
Adipose tissue.
Dr. Carol:
Okay, so I just did a preventing type two diabetes summit thing. And the challenge with insulin resistance is the adipose becomes resistant to the ability of adipose to store the excess calories from sugar. So, insulin makes it possible. I think that’s how it works for sugar to get stored as fat and when the fat becomes resistant to the signals from insulin that says, hey, sugar coming in, turn it into fat and the fat says, no, sorry, I don’t understand you. Norman Fishman developed the protocol for insulin resistance in 2003 or 2004. And he did it by muscle testing or scanning for it. But the literature has actually confirmed what he found. It’s inflammation, trauma. There’s an emotional component. There’s an infectious component. So, he picked 230 and 430. And since then, I’ve updated the insulin resistance protocol to include 160, just malignant virus and 61. And then, toxicity in the adipose, but inflammation in the adipose. And everybody ran it on when I came home from Dr. Fishman’s place, lost our appetite, lost their appetite for sweets, had their waist size reduced from half an inch to an inch in a week, and they all got constipated. So the first time I taught it, I warned people they would get constipated. And one of the Naturopathic muscle tested his staff and said the toxicity goes from the adipose to the parasympathetics. So that fixed the insulin resistance protocol so it didn’t make people constipated. But then Rob DeMartino gave a lecture about leptin resistance, and it’s too complicated to do without slides, but leptin controls insulin. Leptin and adipose go together.
Dr. Carol:
The Vagus is involved, stress is involved, melatonin is involved and leptin sensitivity. So leptin resistance, insulin resistance, leptin sensitivity, insulin sensitivity, those go together. You have to watch that section of the Advanced. But that’s the key. So somebody that’s 50 pounds overweight, the problem is not so much the weight, although that’s a problem for joints and blood pressure and all that. But the adipose is inflammatory. It’s not just a storage site. It’s actually immunologically active. And that inflammation and the constant need for insulin basically exhausts the pancreatic islets and you get type two diabetes. Because you’re overweight, but not just because you’re overweight. It’s because the pancreas has to keep producing more and more insulin to drive the excess calories into fat. It’s complicated. It makes my brain hurt.. It’s even worse when you read the slides because it makes sense when looking at the slides. But the thing that we have as an advantage over anybody else that’s treating this is that we can treat the Vagus.
Kim Pittis:
Yeah.
Dr. Carol:
So the vagus nerve tells the liver to put out less sugar. And the vagus nerve tells the brain don’t stress, dude. It’s fine. We’re all good down here. So, you put those three pieces together, and it gives us an advantage. Leif Erickson says Harry van Gelder quote, Always ask why. That’s where I got it, right? I lived with George for 31 years. That means I lived with Harry van Gelder for 31 years in my head.
Kim Pittis:
Yes. Why? It’s like when my kids were toddlers and it’s we’re going to go now. But why? Because it’s time to go. But why? Put your shoes on. Why? So I’m a middle-aged toddler now, asking my patients. Okay, but why?
Dr. Carol:
Just listen to your inner child, right?
Kim Pittis:
Yes.
Dr. Carol:
All right.
Kim Pittis:
Derek. Oh, Derek, I’ve been trying to email you, but Derek says get the emotional aspect out of the way first. That’s a big component. And that’s, again, a reason to have multiple machines, right? Like to be able to address the emotional side of everything while dealing with the scarring or the inflammation or the torn and broken and the bleeding is huge.
Dr. Carol:
And when you have a patient where they have multiple things going on, I have one CustomCare that’s concussion in Vagus that runs. That’s 47 minutes, and I have 60-minute appointment slots.
Kim Pittis:
Yeah.
Dr. Carol:
And then TTH.
Kim Pittis:
Yeah.
Dr. Carol:
Nobody should have this many things that happened to them in the last 18 years. This is not normal.
Kim Pittis:
Yeah.
Dr. Carol:
So THH also includes the frequencies for the emotional component. All of the emotions are in order.
Kim Pittis:
Yeah.
Dr. Carol:
Terror, overconcern, anger, resentment, then grief.
Kim Pittis:
Yeah.
Dr. Carol:
And then 970 and 9 life has lost its sweetness and then restoring joy, that kind of stuff. Well done, Derek. Thank you.
Kim Pittis:
Yes.
Dr. Carol:
And Leif, 124 and 710. It works for lumbar vertebra, but the thing that everybody needs to understand and the challenge we have with some patients is they are used to passive treatment. I go to my chiropractor twice a week. I get a massage twice a month. And what are we doing for exercise? It hurts too much when I exercise, then we’re doing the wrong kind of exercise. So, there’s no way for passive treatment by itself to fix you. I can get you out of pain. And these are the exercises you’re going to do to repair the disc.
Kim Pittis:
Right.
Dr. Carol:
Or the facets.
Kim Pittis:
Right or the bursa or whatever. How many patients did I have this conversation within the past two weeks about getting an epidural or a cortisone shot and thinking they’re done now with me? I’m like, No, I’m so happy you’re out of pain. And that injection got you out of pain like that. But now the work gets to start because now that you’re out of pain is your opportunity. Now is the window to go in there and get things firing, get things structurally where they’re supposed to be.
Dr. Carol:
Stable circulation, proper muscle contraction and I’m stepping into your sandbox here but proper muscle function brings circulation. I’ve had blown discs in my low back since 1996 or 1997?
Kim Pittis:
Yeah.
Dr. Carol:
And the disk was thin and dark on an MRI, and I ran Microcurrent and I did the exercises from new heights, contracting the multifidi and the rotators. And five years later, when I did something else traumatic, I tore my SI joint, but they did another lumbar MRI, and that same thin dark disk was now thick and white and fluffy because I did my exercises for six months and corrected my posture.
Kim Pittis:
Yeah. There’s another good quote. The first thing to do when you want to bail out a boat is to stop shooting holes in the bottom of it.
Kim Pittis:
And I laugh because it’s common sense to so many of us, but not to people that are in it and have never been told another way to do things. I had a patient this week, new patient, who you’re going through. You’re asking why about the pain? When does the pain get worse? When is it better? And he was like, oh, it’s so bad when I wake up in the morning, I’m like, okay. And he’s like, Why are you smiling? And I’m like, Because your next thing is going to say. But as I get moving, it improves. And he’s, yeah, I’m like, fantastic. He’s like, What are you talking about? I’m like, Movement makes the pain go away. That’s great. This is like easy.
Dr. Carol:
And then the next question is, what position do you sleep in? I sleep on my stomach.
Kim Pittis:
That’s exactly how it went. Right. And people who are stomach sleepers, they end up looking, I always say it’s like Spider-Man crawling up the wall, like everything is rotated and twisted. And then he’s but my aura ring tells me like I barely move. And I’m like, That’s even worse because you’re stuck in that position for eight hours. So everything is torqued, rotated, stacked,
Dr. Carol:
Pressed,
Kim Pittis:
Pressed, compressed, fractioned. So it’s all about reframing it, I think, right? So asking those questions.
Dr. Carol:
Framing is a good word. Hey, Derek needs to know if you’re going to stay at his place.
Kim Pittis:
Yes, I’ve been trying to email him about that. So Derek, if you can call me or email me, we can discuss it off the show. Nina asks, what do you suggest for a client with a benign cyst on her sternum and ribs?
Dr. Carol:
Number one in my experience, 59 never works. I’ve never had it work on a cyst. It seems to at least I have the hope that it will reduce the tendency to make cysts in response to inflammation and toxicity.
Kim Pittis:
Okay.
Dr. Carol:
A patient with a four-centimeter breast cyst and we went to the imaging center. They did an ultrasound of her breast, identified the size of the cyst. Then, we went back in the dressing room and I treated it and with my hands on it, frequencies for cystic condition did zero. Frequency for inflammation reduced it. The frequency for toxicity took it down by 25% in, I don’t know, 20 minutes. So, it was 30 minutes between one ultrasound and the next one. And the volume of the cyst was reduced by 25%.
Kim Pittis:
Wow.
Dr. Carol:
Her Ob/Gyn still went in five days later and drained the cyst, but it never reformed. So the cyst is coming from someplace. So, treating the cyst. I don’t think Nina is the thing. And if it’s on the sternum. Derek, you’re right. It’s 783. But the cyst forms in the superficial tissue between the bone and the skin, which is fascia.
Kim Pittis:
Yeah.
Dr. Carol:
And if you look at the lymphatics, maybe that goes down the sternum. And then when did it start?
Kim Pittis:
Yeah.
Dr. Carol:
And the other thing is if you can’t change it, have a dermatologist drain it. And treat it for wound healing. FSM is not the solution to everything, so have somebody take it out. And then treat it.
Dr. Carol:
For wound healing and you’ll be better in 2 or 3 days. There’s no general anesthesia. It’s a local.
Kim Pittis:
Yeah.
Dr. Carol:
It’s either 1 or 2 stitches or just a Steri-Strip depending on the size of it.And you have to find out what’s in it.
Kim Pittis:
Yeah.
Dr. Carol:
Is it truly benign? Who knows it’s benign unless they biopsied it. How do you know it’s benign? Because somebody said it was benign. Great! How do they know? I muscle-tested for it. Don’t get me started. So, there’s that. So way to go, Nina.
Kim Pittis:
Great. I’m asking about FSM’s in Great Britain. I’m sure on the website.
Dr. Carol:
Eva and her team are in London, the northeast part of London. I can see her face. And she’s in Bath. One of our instructors blond hair, brown face. I can see her, so I can’t remember her name, but we have quite a few practitioners now in the UK because we’ve been there for a couple of years and in Ireland as well. Brid Hanlon, she took the FSM course, but she’s really a fan of the Healy and the Mag Healy. She took the FSM course, I think, the first time we went to London when we went to the osteopathic college because the lady who’s wonderful, Angela Stephenson, thank you, is an D.O. And so the first course we did was at the Osteopathic College near London Bridge.
Kim Pittis:
Oh, and Debbie Beighton says that she’s in Sussex.
Dr. Carol:
East Sussex. Yeah. She goes, Oh, Debbie. Hi, Debbie. And then Brid is adorable. She’s fun. You just love the disbelief on your client’s face when they can move something they haven’t moved in months or years. Isn’t that the truth?
Kim Pittis:
Yeah. There’s nothing that quite whatever replicate that when somebody. Oh, you’re reading something interesting. Hang on.
Dr. Carol:
And the lady with the CRPS, she hasn’t been able to talk in a year and a half. And at the end of the hour, the pain is gone. And I said, okay, try talking and she was afraid. And I said, try it. She was able to talk and then it stayed. She was able to talk. She was able to eat crackers and swallow for the first time in three years. She’s at least 20 to 30 pounds, underweight, because she hasn’t been able to eat. Without pain. It’s pretty cool. Anemia.
Kim Pittis:
Oh, where’s that? Justin’s for anemia. Okay.
Dr. Carol:
Hi, Margaret.
Kim Pittis:
That’s all you.
Dr. Carol:
I’d find a different hematologist. But something’s not making sense. Iron supplements do nothing. So lack of iron is not the problem. What is the problem? That’s yeah, the hematologists and endocrinologists. I’m not entirely sure what the problem is in that arena, but a patient that has an empty sella and the endocrinologist won’t test signaling hormones. Why? Because the signaling hormones come from the pituitary, and the place where the pituitary should be is missing on this patients. So why would you not test signaling hormones? I don’t get it. I don’t understand. It’s not my scope So there’s got to be somebody out there. I’m glad you agree, Margaret, and I don’t know what the solution is. If iron doesn’t fix and then iron is not the problem. Two blood transfusions and she’s not bleeding, 18/62. Okay. I give up.
Kim Pittis:
Yeah.
Dr. Carol:
Onward.
Kim Pittis:
Onward. So, circling back to my hip flexor people, because I do get these questions sent to me quite often. And I want to simplify something as best as I can for people. So everybody’s going to come on a little journey, a little story with me. So our anterior chain, like I said, so everything on the front of us, patients who are listening, I want you to pay attention to this also. We’re chronically shortened because we sit too much, we drive too much, we don’t take breaks so the muscles on the front of us tend to just… you’re never going to have like a foldy muscle, like our body will adaptively shorten to take up the slack. So like the hip flexors get affected by far the most because they’re always scrunched up because we’re sitting in these chairs and then the front of us, like our abdomen and our pecs and everything shorten as well because we’re typing and cooking and driving and we’re close together. So when that happens, the people who are listening on the podcast can’t see my fingers but imagine like my fingers are interlaced and I’m pushing my fingers together. So when our bodies are adaptively shortened, our muscle fibers like glued together and we’re going to go back to the sliding filament mechanism or actin and myosin, those heads can’t contract properly because they’re overlapped. So that’s what we call there’s a stretch weakness and then there’s a shortened tight weakness. So just because something is tight like the hip flexors or they’re adaptively shortened, they will test weak because the actin and myosin heads that have to contract with fiber, they can’t optimally grab each other. They’re all shortened up in this bundle. So when we’re talking about frequencies to help with that, I found that 13 obviously so scarring because 13 is that magical frequency that helps unleash or helps relax or vibrate apart those bonds that hold the fibers together. So when those fibers now have the permission to not be glued together, they’re able to create space. And then it’s like taking a breath, right? Oh, I don’t have to be stuck to my neighbor anymore. And then when they’re in that optimal position, then they’re able to contract properly. So 13 is and 51, 3 and 97, like all those like scarring fibrotic type of frequencies are going to work really well. But then you have to follow that up with increasing the secretions to the area. Because if those fibers, if those sarcomeres have been like bound together, they need to have increased secretion so that they can move apart optimally again.
Dr. Carol:
And the other thing is that if you’ve been stuck this way, flexed, the muscles in the back are inhibited because the muscles in the front are tight.
Kim Pittis:
Exactly.
Dr. Carol:
After you loosen up the front, the next step before they walk out of the office has to be to get them to sit up.Straight, keep their chin in neutral and take their shoulder blades and activate if the only thing they ever do is activate the lower trapezius.
Kim Pittis:
Yes.
Dr. Carol:
That seems to let the whole posterior chain know, Oh, you want me vertical. Oh, and now that the front isn’t tight, I can contract. What a concept.
Kim Pittis:
What a concept. So, to piggyback on what you were just saying, the opposite of that tight weakness is what we call a stretch weakness. So the Erector Spinae group in the back is the rhomboids. Like I said, the biggest thing that makes my head spin around in a circle is when someone says that a patient has tight rhomboids because nobody ever on the planet in 24 years has had tight rhomboids. They have slips of the rhomboids that have contractures and trigger points in them 100%. But a rhomboid is not ever tight because we’re never in retraction.
Dr. Carol:
And the thing that’s tight in between the shoulder blades is the longissimus thoracis and cervicis that are right under the rhomboids. And it’s easy to get suckered into thinking it’s the rhomboids. But when you feel it, the thing that’s tight doesn’t run laterally or at an angle. It runs vertically.
Kim Pittis:
Yes.
Dr. Carol:
It’s beneath the rhomboids because I don’t know what your cadaver look like, but my cadaver was a well-developed adult male and his rhomboids were thickness of two pieces of typing paper.
Kim Pittis:
Absolutely.
Kim Pittis:
They’re insignificant. I don’t want to say insignificant, but they are not these massive muscles that people give them so much credit for. Like, I get it. You point on it and some massage therapist or some trainer said, Oh, those are your rhomboids because the fibers look like they were running like this. But that’s not always the case. That’s never the case. So, going back to those muscles that are chronically elongated. So, that’s stretch weakness. So the back muscles because we’re hunched over, the rhomboids because they are in this protracted state. So they are going to respond very well to 124 because you’ve got these micro teares in them because they’re elongated. So these fibers are pulled and stretched apart that’s torn and broken. So running, 124 first can be really helpful because that’s the cause of the pain is because they’re torn, they’re elongated.
Dr. Carol:
And the muscle itself may not be why they’re painful or weak. But if you look at the anatomy, open up Netter, and they connect to the scapula or the spine with a layer of line of connective tissue. So it’s 124/77 the connective tissue. 124 in the fascia.
Kim Pittis:
Yeah.
Dr. Carol:
In order for a muscle to be strong and functional, it can’t go from contract, contract. It has to relax and then contract.
Kim Pittis:
Exactly.
It can’t go from eccentric stretched if it can’t. So it has to move.
Kim Pittis:
Right.
Dr. Carol:
Fractions.
Kim Pittis:
That’s right. And to your point, again, it’s the agonist-antagonist relationship. So if our primary mover or agonist is our Core, the antagonist is our back, both have to be on board with that. This movement is going to happen. It has to be perceived as being safe and functional for both sides of it to happen. So, we’re not going to get into this great extended posture if we have adhesions in our abdomen that’s perceiving extension as dangerous, we have to give permission for these muscles to elongate for those muscles to contract. It’s like a yin and yang.
Dr. Carol:
And the advantage that we have with FSM is the ability to treat adhesions in the nerve and reeducating the cerebellum, the thalamus, because if it has been chronically painful, the thalamus… Everything goes through the thalamus.
Kim Pittis:
That’s right.
Dr. Carol:
Ascending. Descending. The thalamus gives permission. If the thalamus says, nope, not going to do it. Doesn’t matter.
Kim Pittis:
Yeah.
Dr. Carol:
Quiet down the thalamus. You turn on the cerebellum, increase secretions in the cerebellum, but then you have to increase secretions in the sensory and motor cortex, so the sensory and motor cortex can find it. Now that the thalamus is not standing. So if you think of the pathway, there’s a cerebellum, the thalamus and the sensory motor cortex.
Kim Pittis:
Yeah.
Dr. Carol:
So if the thalamus is saying, no, you’re not going to move it, the sensory motor cortex never gets to send the signal to the cerebellum that says, Hey, sit this way. Not that way.
Kim Pittis:
That’s right.
Dr. Carol:
So first thing you do is quiet down the thalamus. Then I always, I might be wrong, but I turn the cerebellum on first because the cerebellum coordinates movement, and then the sensory and motor cortex initiates it.
Kim Pittis:
Right.
Dr. Carol:
You go sense thalamus, cerebellum, sensory, motor cortex. cerebellum again.
Kim Pittis:
Yeah.
Dr. Carol:
And it gives us an edge. So you can do in 60 minutes what would take a month or two with just exercise and postural retraining?
Kim Pittis:
Yeah.
Dr. Carol:
Because if the brain is afraid to do it.
Kim Pittis:
Yeah.And that’s why I love the second day of the sports course is because that’s all we do is and when we had it at the facility in Troutdale, we had this practitioner, she was an optometrist, but elite judo athlete But it was so interesting because she had this knee injury, but because of her judo, she was always on her toes and knees over toes and just hunched and ready. And I’m like, we need to get your posterior chain firing because you’re all anterior. Everything is on your toes. And a lot of athletes love to be in this, like sympathetic. The tiger’s coming. I got to get ready to run as fast as I can. No, getting you and so sometimes, like with her, it was a simple cue of saying, I want you to put your weight on your heels and it was like you could just see it in real-time. You could see the hamstrings engage, the glute engage, the lower back engage, the lower traps turned on. But we did have to take all those steps that you were just saying because after 40 years of doing one thing up on your toes, it’s not going to just resolve and turn around overnight. So we get to help it. And after 20 minutes, she changed all of her mechanics because that posterior chain was like, Oh, this is safe. There’s not a tiger coming right now. Cool.
Dr. Carol:
And judo and most of the martial arts are all forward.
Kim Pittis:
Yes.
Dr. Carol:
And it’s combat.
Kim Pittis:
Yes.
Dr. Carol:
So the thalamus, the limbic system is part of the training in the sport.
Kim Pittis:
Yeah.
Dr. Carol:
You have to prepare for combat for sure.
Kim Pittis:
but just standing and waiting for your Uber, it’s not it needs to be engaged.
Dr. Carol:
Did you see Rick Allen’s suggestion.
Kim Pittis:
About doing a frozen shoulder thing? That sounds cool.
Dr. Carol:
So we could do like a two-day thing in Troutdale if you’ll come up.
Kim Pittis:
I would love that.
Dr. Carol:
Wouldn’t that be fun? And just do shoulders?
Kim Pittis:
Oh, yes, please. Okay.
Kevin:
And is Rick Allen the one that does our theme song?
Dr. Carol:
Rick Allen? No, that’s Rick Koi.
Kevin:
Rick Koi.
Dr. Carol:
Rick Koi does things.
Dr. Carol:
Rick Allen is one of the long term.
Kim Pittis:
Awesome. Thanks.
Dr. Carol:
Okay. Scleroderma.
Kim Pittis:
Oh, we’re almost at the time is beeping, but let’s read Debbie’s really quick.
Dr. Carol:
Okay, so scleroderma was, 3 or 4 years ago when Walter presented the case report or I presented Walter’s data. Friend visiting this weekend. I’m going to treat trauma before, bilateral pneumonia, her heart race. So she had to have her heart reset. Scleroderma, it’s an autoimmune condition that affects that creates inflammation in the capillaries, that causes scarring, especially in the fingers and forearms. It’s an autoimmune. So you have to turn, do concussion in Vagus, turn the Vagus on to stop the autoimmune condition. But then you treat scarring in the capillaries, the connective tissue, the joint capsule, the fascia, the nerves, and you run it from neck to hands and you do manual therapy, but you have a second machine running on concussion and Vagus for the whole hour and the data on improving the cold chain hand scores was just extraordinary. Like they went from not being able to pick up a coin to being able to pick them up. It was amazing.
Kim Pittis:
Amazing. Rick Allen, great. See you soon. Yes. We’re going to get on this.
Dr. Carol:
This is so much fun.
Kim Pittis:
So much fun. It was so great to be back. And again, we haven’t really gotten through all of my list, but that’s okay. I’m patient. Everybody enjoyed my Q&A with, we had Dr. Jen Sosnowski on and I made her.
Dr. Carol:
Oh did you?
Kim Pittis:
I brought Jen on again and I had her do a functional medicine talk more like for patients because I have a lot of patients that listen to our podcast. And they were like, she was fantastic, but I didn’t understand anything. I’m like, because it was very much practitioner-based. So I had her like, do like a webinar on functional medicine just for patients And even that got pretty intense because she.
Dr. Carol:
You get in the weeds because the more you know the more you know you don’t know.
Kim Pittis:
Yes.
Dr. Carol:
And the basic challenge with functional medicine is everything is connected to everything.
Kim Pittis:
Yes.
Dr. Carol:
There’s that. And the gut is the second brain.
Kim Pittis:
Yes.
Kim Pittis:
Yes.
Dr. Carol:
The liver and the Vagus and the Vagus controls the senses and controls the microbiome. So everybody talks about the bacteria in the gut and how important that is. And the bacteria are supposed to produce butyrate. You can take butyrate, but being able to treat the Vagus, being able to repair leaky gut and everything’s connected to everything, the gut, the immune system, the endocrine system and the Vagus and the brain. It’s all here.
Kim Pittis:
Yeah.
Dr. Carol:
And it’s magic. I’m so glad you had Jen on.
Kim Pittis:
Yeah, I love our circle of people that I can just be like, Hey, got an hour to hang out with me. 40 of my closest friends.
Dr. Carol:
Yes, exactly. And when I had this SPG-7 person.
Kim Pittis:
Right.
Dr. Carol:
As soon as I got her or before I even put her on the table, I texted Ben Katholi, Dave Burke, and Roger Billica and said, what neurotransmitters? Yes, Gabba! But what am I missing and what supplements do I have her take? In order to… Like if I’m driving secretions in the corticospinal tract, we have to give the cells the substrate that they’re going to be using to create Gaba. What do I give her? And I had text messages back from Dave and Ben, and Roger checks his cell phone about once a week. So, she was gone by the time I got a reply from him. But Dave and Bent said okay, take this. Take that. Only take Phosphatidyl Choline if she’s having trouble with her mouth and stuff that’s coming from the PONS that’s okay.
Kim Pittis:
It is a village.
Dr. Carol:
It’s a village. Like her family.
Kim Pittis:
Yes. Okay. That’s it for today.
Dr. Carol:
4:00 already.
Kim Pittis:
Back to picking cherries.
Dr. Carol:
Yeah. Lucky, you.
Kim Pittis:
All right everybody, thanks for coming. We’ll see you all.
Dr. Carol:
Wait, we’re in Denver next week. Anybody hasn’t signed up for Denver? Kevin is shipping massage tables and table warmers and chocolate today. Be in Denver next week, so you’re on your own again.
Kim Pittis:
All right.
Dr. Carol:
So there you go. It’ll be fine
Kim Pittis:
Has any ideas for me, feel free to email me. [email protected].
Dr. Carol:
Okay.
Kim Pittis:
And we’ll put together something fabulous.
Dr. Carol:
Of course, you will.
Kim Pittis:
Bye, everybody.
Dr. Carol:
Bye.
The Frequency Specific Microcurrent podcast has been produced by Frequency Specific Seminars for entertainment, educational and informational purposes only. The information and opinion provided in the podcast are not medical advice. Do not create any type of doctor-patient relationship and unless expressly stated, do not reflect the opinions of its affiliates, subsidiaries or sponsors or the host or any of the podcast guests or affiliated professional organizations. No person should act or refrain from acting on the basis of the content provided in any podcast without first seeking appropriate medical advice and counseling. No information provided in any podcast should be used as a substitute for personalized medical advice and FSS expressly disclaims any and all liability relating to any actions taken or not taken based on any contents of this podcast.
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