00:36 Understanding the Dura and Craniosacral System
02:53 Anatomy of the Meninges
08:52 Biomechanics and Clinical Implications
33:42 Case Study: Real-Time Treatment
44:33 Learning from Dr. McMakin
44:58 Mobilizing the Neck and Cranium
47:50 Working on the Hips and Pelvis
51:17 Releasing Dura in Sitting Position
56:45 Final Adjustments and Patient Feedback
The Dura and Frequency Specific Microcurrent
[00:00:00] Welcome back everyone. It’s wonderful to have you all here. Isn’t it great that it’s raining today? It makes it so much easier to stay indoors, right? Very excited about our last presentation. Eduardo will be covering the topic of the Dura and our clinical applications and strategies so that, at the end of our day today, we’ll have enough time for everyone to run their things back to their room.
And then there will be a reception tonight. So just reminding you, six 30 to eight tonight coming down, it’ll be a chance to mingle, meet each other, chat more. And so we’ll see you back down here tonight at six 30. Hello, can you hear me? Good evening, everybody. I hope you have sealed some energy for one hour of concentration.
The doura is a big topic. This presentation want to be an extension of what you learn on the course seminar. From Dr. Carol McMakin about the Dura treatment. And as you can see from the title, the cerebral spinal fluid is one of the highest known [00:01:00] elements that are combined in the body. And unless the brain furnishes this fluid in abundance, a disabled condition of the body will remain.
So those are the words of Endot, the father of osteopathy that already understood the importance of biomechanics of the body and of the craniosacral system. After him many osteopaths and being focusing of the understanding, the biomechanics of craniosacral system like Dr. Sutherland, he spent 50 years about to understand the relationship between the temporal bone and the sphenoid bones.
But furthermore, we got viol of Ryman. That in 1971, she developed a special machine to detect the craniosacral rhythm. And this machine, it looked more like a torso machine. The patients had to screw tight in its head and holding the breath for more than one [00:02:00] minute, but she was able to demonstrate that a third rhythm exists in our body.
But in 2000, with the computer and with the special machines, we demonstrate that this movement exists and we have this nice diagram that shows up how we have a flexion and extension with the steel point in between in the craniosacral rhythm that is happened in the brain, in the cranium and in the wall body.
And here we have a nice animation that shows how. The three rhythm, the heartbeat, the respiration, and this expansion and retraction of our C central nervous position happens.
So the topic here are the meninges. Today we have three layers of meninges [00:03:00] from inside out, we have got the pi matter, the arachnoid matter, and the dura matter. Those meninges want to protect our central nervous system, our brain, our spinal cord. As we can see, we got the dura matter, and then there is a space between the dura and arachnoid that is a subdural space.
The arachnoid matter and P matter forms the lepto meninges and in the space between the arachnoid and P matter is a subarachnoid space that contains CSF major vessel and all the cranial nerves. As we can see, the dura is divided in two layers, the industrial layers that are covering the inside of our skulls and this exit the foramen magnum and then it became the periosteum of the outside of the cranium.
We can see that there is no [00:04:00] space between the industrial layer and the cranium, but it creates space. When we have some bleeding of a artery and it can form an extradural space, that is a path pathological space. We see that. We have the industrial layers and we have the inner menal layers. They are always fused together, in exception, where they form the spaces for the dural venous sinus and where they form the dural en foldings.
We know that the inner meningeal layers will cover all the spinal cords and it’ll end at the level of S two vertebrae. So we see here the main menal. In foldings. We’ve got the biggest one that’s far Cerebra, that’s the largest one, and we have tentorium, cere belly, and Far ce. We see that the forks is the touch on the front of the cranium with the crita galley, and on the back of the [00:05:00] cranium in the internal occipital protuberance.
Then we have the Tanium that divides the cerebellum from the brain, and he attach on the backs of the cranial and the turn opposite Cs. In the past glenoid process, and then the groove of transverse sinus and superior parts of petro part temporal bone and on the frontal part is attached on the glenoid process.
Then we have the diaphragm of the cellica that is on the top part of the sphenoid bones and it’s covers our pituitary grants. And so we see how it’s important, this one for our endocrin systems. And take a look how many structure are passed through this bones. We got cranial nerve 3 4 6 V one V two, and we have internal Caro [00:06:00] artery with the post sympathetic fibers.
So real important space, and we have now the possibility to work this space with the fsm. We see that the dural fuse, we have the subarachnoid space and the subarachnoid space will give some arachnoid granulation that we gonna let the CSF pass from the subdural space to the dural sinus and they will drain to the inter jugular veins.
So this is the cranial fossa, and we can see how the dura is innervated in the different part of the cranial fossa. On the frontal part, we see the, it’s innervated by the all three branches of trigeminal nerves. The middle cranial fossa from V two and V three, we have the infratentorial fossa that is [00:07:00] innervated by C two and C3 and the vagus nerve.
And then we have sartorial dura that is innervated by ophthalmic V one. And from here we can understand all the referred pain from dural inflammation that we can have in the face, in our eyes, in our neck, in our shoulders, due to dose information. We can see here the vascularization. We have the middle meningeal artery that is the branch of max artery, and it takes.
Vascularization from the frontal part and from the middle part of the cranium. We’ve got this part here it called, and this the softer part of the bra of the skull. And so when we had the accident, this is the part of the skull that is more allowed to a fracture.
Sorry. [00:08:00] So we can see that all the dura in foldings are followed by dur venous sinus. Take a look how many sinus we have in all part of the brain. And so how important is for the dura meninges to work in a proper way to let the drainage to all the part of the brain to do in the proper way
in the center of the brain, we add four ventricles to measure one. One on the left, one on the right. Then we have the third ventricle and the fourth ventricle, and then we have the central canal. On the ventricle, we have got the current plexus that allow the production of CSF and the storage for CSF that the brain needs.
So we go a little bit on the biomechanics. We know that RCP miner is the only muscle of the suboccipital muscle that [00:09:00] its connect directly to the dura. So try to imagine a dysfunction of the upper neck vertebrae, what can cause of traction in the doura. And then we see that the dura is attached to the cranium, to the atlas axis, to the vertebrae, and all the dura sac can be attached to the exit of the nerves.
And then down to the second. Sacral segment and to the field’s terminals. But what do you think, what can cause this function on this part of the neck? What this part of the neck is related to? If you can close your eyes and touch your upper neck and just move your eyes little bit on the left and little bit on the right, and you can see the activation and correlation between the [00:10:00] reflex that we have from the muscle of the eye, and.
The muscle of the upper neck. And so we see that we have got visual system and neck muscle. They are correlated by the ocular motor muscle are essential to perform a combinated eye and neck movements. We have got the core reflex, the OS reflex, and we know that those muscle can let our body to adapt for the disorders of refractive systems that we have myopathy, ime, and the stigmatism.
And then as you listen at previous, on the previous presentation, we got our SOO system with trigeminal nerves that there is connected to our upper cervical. To the trigeminal nucleus [00:11:00] Alis C one, C two, C3. And so all the dysfunction of the TMJ can be related to this dysfunction of our upper neck and v versa.
So all this can be on the little bird that can let us try to work on the dura. So a little bit of anatomy. We know that each bones can move and so we have a maneuver for each bones, but we don’t have time to cover this. And between two bones we have a sutures and we know that the sutures must to be soft when the sutures is in this function.
It can be painful or can be really hard when we touch. And so this is something that can let us to understand it. We have to work. That’s structures. But if we know anatomy. We can understand how important is our upper neck [00:12:00] protocol. We see on the forme Magnum that we are cranial nerve 11. Of course, we have got the brainstem with the artery and other vessel.
Then we have the Jura foramen, and it’s the exit for cranial nerve nine, 10, and 11. So think how much is important, those little hole for all the function of our speaking, swallowing, and for all the things that vagus nerves do. And so we can’t miss the structures if we want to be or give an influence to the biochemistry of our body or to the biology of our body.
We always have to start from a structure. We see that carotid canal with a car, internal carotid artery, and we have [00:13:00] the stoma foramen for the exit of the fascial nerves. And so we can maybe pay attention for all the problems about fascial, about ears. And here we got the orbital cavity and pay attention of what we have inside of those bones that are construct the cavity of our eyes, but got cranial nerve two, cranial nerve three four V one V two artery and veins.
So if something is not going well with one of those nerves. Maybe we can try to fix the structure. Firstly, try to release the Dura addition and let the bones move how they have to do, and maybe we can solve some of the problem. [00:14:00] We see the forum and und with the V two and see how the V two is going to the orbital cavity and to the maxillary bones.
How many of you I got some problem to work on trigeminal nerve with the patient? How many patients have headaches problem behind the eye? And then we got the oval hold with the passage of V three of mandibular part of the trigeminal nerves. Now we have got some really nice picture on cadavers. Look at the dura, how it protects all the brain.
And on the second image, we can see all the artery that are glued together with the dura.
We see here the [00:15:00] skull, the external part being removed, and we see how the doura is inside. With the artery, we got the arachnoid, but in the DY body, there is no CSF, so the arachnoid is down on the Pia mother. And here you can see just on the center, the P mater, the arachnoid is taking off from that part.
And you can see that the P mater is like the skin of our brain and is protecting our brain in his entire surface. And we see that the p mater is attached to the glial end fit and is not possible to remove the P mater without damaging those cells. Here you can see the how the meninges R in the [00:16:00] real body and see how sick they are and how strong they look.
So even when we do our cranial manipulation, we have to be gentle but not so soft. Sometimes we have to push a little bit hard if we want to move a membrane like that.
And here you see the division between the two dural unfoldings between the vessel that are passing through there.
Here we have all the spinal dur matter, and you can see the ponds. Upstairs and then we go down and we see all the nerves of the enlargement of the spinal cord, the brachial plexus, and down how the level of L one, L two. It becomes a coda, a equina, and it end up with the film terminals we see here inside of [00:17:00] the spinal canal, how the, it’s attached or the spinal cord or around here we have the ventral view of the cervical enlargement of the spinal cord and see how the nerves is passed through the dura and how they are really melt together and try to imagine attraction in the dura.
What can cause of the nerves ending that we have there? Here we have the dorsal view. What is nice, you can see that tiny spider web looks like that is the arachnoid.
And here we have got the Kai Equina with the fill terminal, and we see how the fill is attached to the co coys and how the coys, if [00:18:00] it is in this function, can create a tension on the dura or vice versa. A dura can really increase the tension on the Coxs. And so when we have a Cox Nia, sometimes we don’t have to check the Cox where we have to check the cranium, the TMJ, the suboccipital muscle, and what’s going on in that part of the body
here, the arachnoid in the cervical region. Here we got the arachnoid in the dorsal region, and you can see. How a ganglion is protect inside between one vertebrae and another one
here I’ve got with spinal meninga dissection of the cervical spine and we can see the cerebellum and how the meninges are protect that part of the body. [00:19:00] And here we can see the accessory nerves that we work with. 40 94 when we do our upper cervical treatments and we do the C one and C two nerves that are passing that part.
And here we got the thoracic. And in yellow on the right side you can see a ganglion. And then with the two roots, the dorsal and ventral roots are dividing. Here we have the sinus, the sagittal sinus, and the sinus of the tentorium. How the dura, it’s covered them. Here we got posterior view and we can see the faults and the tentorium and how those meninges really give the right space and divide the brain.[00:20:00]
The suture of the cranium should be soft, but they can get ossified or remain movable depending about the force that they’re gonna receive. From our posture and from the biomechanics of our spine. Here we have three animation about the craniosacral expansion and retraction. What happened in our skull call with flexion and extension, and we have the movement between the sphenoid and the occiput.
So all those are moveable structures and they can be dysfunctional and we have the possibility to work on them. And the same how we got the rhythm between the cranium [00:21:00] and the sacrum, and how everything in our spine must be movable to let our nervous system do his works in a proper way.
Here we go. So brain in motion. Let’s have a look on each heartbeat, what’s up in our brain, and see how the heart is connected to the meninges of the cranium and of the brain, and how they have to move properly to allow this rhythm to propagate from the chest to our brain. So we discover the neuroscience discovered that during the Ram State, our brain volume reduced [00:22:00] drastically to allow more CS, F to go inside the brain and take out all the waste product and give some nutriment to the cells.
This is an MRI that the neuroscience did during the REM state. Unfortunately, the patients have to sleep inside the MRI machines. I dunno how they did it. But anyway, they do the study and so we see how important is the sleeping part and how it’s in correlation with the good movement and biomechanics of the meninges.
So the lymphatics and meningeal lymphatic system are critical for the homeostasis of the central nervous systems, and we can see that. If we are deprivate from sleep and we have got reduction of the re state, that can cause deposition of insoluble amyloid B, and that is followed [00:23:00] by intracellular aggregation of tau in the narrow cortex and subsequent neuro cell loss.
All these are related to lots of neuro neurodegenerative disease like Alzheimer, systemic lupus, multiple sclerosis, rheumatoid arthritis, grogan syndromes, Hashimoto thyroiditis, and many more. So here we have a little video. It’s two minutes that will explain and show how the glymphatic system works and so what we have under our hands when we are touching the head of our patients.
This is a brief overview of the glymphatic system and cerebral spinal fluid flow. The glymphatic system is a fluid transport system that accesses all regions of the brain. It has three parts. Part one is the influx of cerebral spinal fluid portion. Part two is the exchange of cerebral spinal fluid with interstitial fluid of portion, [00:24:00] and part three is the flx of interstitial fluid.
Cerebral spinal fluid is produced in four interconnected cavities deep within the brain, known as the ventricular system. Within each cavity is a choroid plexus, which is comprised of capillaries encased within append themal cells. Cerebral spinal fluid is produced in the ventricular system, flows in the subarachnoid space, which is the space in between two of the three membrane layers, encasing the brain and spinal cord flows by way of the glymphatic system into spaces around cells where it provides vital nutrients to the brain cells.
Blends with interstitial fluid. Interstitial fluid flow then picks up waste or potentially toxic substances flows by various routes to drainage pathways back to systemic circulation. [00:25:00] This slide represents a detail of the brain tissue. The pink cells are astrocyte. The green cells represent neurons in synapses.
The space around cells is known as the extracellular space, also known as the interstitial space, which is filled with inter with interstitial fluid and extracellular matrix. The bold red and blue lines represent vasculature. The red represents arterial inflow. The blue represents venous outflow. The blood vessels are encased in astrocyte end feet, and the astrocyte end feet leave a little space in between the inner surface of the end foot and the outer surface of the blood vessels, creating what’s known as a perivascular space.
There is a perivascular cerebral spinal fluid influx pathway, a perivascular [00:26:00] interstitial fluid, F flux outflow pathway. But black dots represent waste and potentially neurotoxic substances. Cerebral spinal fluid flows in the perivascular influx pathway to flow in between or through specialized channels within the astrocyte and feed, to then blend with interstitial fluid and the flow of interstitial fluid then disperses the vital nutrients to the brain cells.
The flow of interstitial fluid also helps to pick up waste and carry them to the flx. The outflow pathway where those substances as carried by interstitial fluid will flow through or in between astrocyte mfe, so through the mfe, within specialized channels to the outflow pathways. And then through various routes back to systemic [00:27:00] circulation.
So this is showing inflow through or in between. The end feed blends with interstitial fluid. Interstitial fluid flow will then help to carry waste or potentially harmful substances to the outflow portion, to then flow by way of the outflow to various routes back to systemic circulation. A disturbance in the glymphatic system can lead to a lessening of nutrient flow to brain cells, as well as a buildup of waste and neurotoxic substances, which over time can lead to neuropathology.
So this has been a brief overview of the glymphatic system and cerebral spinal fluid flow. Thank you for joining me. Here we had a nice explanation of what is happens on each cranial rhythm [00:28:00] in our brain, and we can understand now the importance of the astrocyte cells and what kind of function they have.
They support and structures, they help blood, the brain barrier mountains. They are really can give a correlation of nutrients and loss of homeostasis. They regulate neurotransmitters repair and scar formations. A communication between neuron. So when we have a cell injury on infections, we have a microglia that will act, they know the microglia, it’s like the immune system of our brain, and they can lead of the activation of inflammatory response and acute inflammation.
So the serious causes of dural addition are rare, but in more common and likely to affect, many of us in a certain [00:29:00] degree, is the formation of scar tissue in three places between the layers of meninges themself, the meninges and bones and spinal column, and meninges and the spinal cord. So those are all the structures that we can work when we analyze.
The postures and biomechanics of our patients, and we can add even sclerosis from adipose tissue because we know that internal part of our spinal canal are covered by adipose tissue. So the dura is interconnected with the autonomic nervous systems, and it can have some correlation with the vascular regulation, pain, modulations, autonomic reflex, cerebral spinal fluid regulation, and autonomic dysfunction and headaches.
So we know that the dura and fascia, they’re really [00:30:00] interconnect when the nerves it’s come out from the spine from. The first part is still protect from the dura and then it melts with the fascia already. That is related to our vertebrae column. And so if a fascia is pulling, the fascia can pull the nerves and the dura.
And if the dura have a dysfunction, it can pull the fascia as well. So you see down on the spinal cord and will dural and fascia restriction how the spinal cord can be altered in its form. And here we have another little video and we can see how a dysfunction in the fascia can lead to dysfunction in the spinal [00:31:00] and in the dura through all spine.
The same can happen in a shoulder attention, and the fascia can give attention in all the neck and the meninges of the neck.
And lastly, we can see how upper cervical dysfunction can give attention in our brain.
So there is a nice study that relate the posture on how a mice sleep and the production of CSF. F and we can deduct that. The posture of our body and the posture of our head in the space. It’s correlated to the right amount of the CSF that we can [00:32:00] produce and alteration in our postures can relate it to malfunctioning of production of CSF and of dural addition.
So we said before that the dura is interconnected with the fascia, and we know that the fascia and muscle are divided in seven myofascial lines, and we know that the dura is interconnected with the superficial back line, with the deep front line, superficial front line s spiral line and lateral line. So the dura can involve in the postural imbalance and vice versa.
So the CSF is important. Before that, we board. At the moment that we board, when we start to become an adult and we see that, then there is a moment where if the [00:33:00] CSF is not produced in the right way, so this means that the DME system, the craniosacral system, are not working properly, we can go to the neurodegenerative disease.
And with the FSM, we can really invert this status and go to the normal aging. So we have the possibility to do that. So dual addition surgery may be that in the close future, FSM will use instead of surgery, of course, when it’s possible. And so FSM is a hope for the future of medicine. And if you have a little bit of attention, I will show you a case report so you will see a real time treatment on a patients.
These patients came to the core seminar in Rome. It was the patients of one of the [00:34:00] student of the seminar. This guys had a sports injury in 2008 and he hurt his pelvis on the right side. In 2013, the pain was so strong that the neurosurg neurosurgeon decide to fuse L five and S one. He start to develop pain in the middle, back in the neck, in the well body.
So the neurosurgeon decide to remove the implant in 2016, but the pain still get worse. He have has been trying everything. He came in the seminar and I apologize that Carol is not here because it was really difficult patients. So she sit and Carol was analyze him a little bit and so she already immediately understood that the [00:35:00] problem was a si sprain on the right side, that nobody had been treated before.
The pain was centralized and the posture was shifted forward and it create a facet problem and middle back problem. So the patient laid down and Carol put 40 and 10 neck feet. Then we had another machine for facet for the SI joints. So on the pelvis and the on the. On the belly and another machine for the upper cervical working off a set disc.
Then she move the legs. She saw the restriction and she do removing scar from the ureter on both side. Immediately. We know that it’s related to [00:36:00] the functioning of the SOAs and understanding position. It was look like this, so you know that there’s something that is going wrong in that part of the body.
So after 30 minutes, the pain start to slow down for the first time in 15 years and after she moved the legs, she, ben, the legs through the chest. And she saw that the chin was start to jumping out in this way. And so she told me, okay, let’s try to work on the dura. So I was working on the cranium a little bit, something that you’re gonna see here.
She once start to move the hips and the patient start to melt. Then the time was finishing and when he was standing, he was not able to walk in this new body. And so Carol teach him go to the [00:37:00] hills, and that was helping him and try to regain a new way of walking in the new posture that he was missing from 15 years.
And he was so excited by that, that he immediately bought a custom curse. So if a patient bought a custom curse in Italy, first time that he heard about FSM, it means that he had, that the treatment was really excellent. So of course these guys make me a call. Please, can you treat me with FSM? I say, yes, but you will let me do a video.
And this is what we did for you. So as you can see now, in the posture, that unleveling of the shoulders, the pelvis is shifted, it’s forehead postures, and we have the line of the pelvis that is backward in relation of the shoulders and shoulders of back in relation of the head. And you see the bending test, how is [00:38:00] limited, the convergence test.
You see the left eye is not going in the middle as the right one. And we have some computer analysis of postures. So you can see how the gravity line is really on the back and is twisted. So how you start. A treatment if you want to understand if you have a dura restriction, okay. From the history of the patients.
But then we have some maneuver so that now we’re gonna check the dura. We know that the dura is attached to her CP minor to foramen magnum, to the sacrum, to the coxes, and can be an addition all over the spine. So we’re gonna flex the leg nice and slowly and we are gonna see and check if there is a [00:39:00] tension in there.
And there is a tension at this point, and we are gonna see if the chin is gonna move. Of course, you don’t have to push so hard. You will see for fascial restriction. Okay? And we can see. As light movement in the chin. This is an indication for dural addition. So you will see on the chin of the patient this kind of movement more or less so the head goes into slight extinction?
Yeah, because the fa the dura is pulling. So the next maneuver, we are checking the internal rotation and we can see a slight tension in this part.
We are gonna check the other hip [00:40:00] rotation and you see how still some tension on this side, but the movement is quite good.
So the internal hip is the first things that you want to check for dural restriction. Then of course we check a range of motion, are checking the range of movement of the head, some tension here on the right. Nice and relax. We work with three machine and dura, some tension cord still here on the left and 3 97 3 sclerosis and adipose tissue.
I have my finger on the suboccipital muscle. I feel that RCP minor, the only muscle that is attached to the dura is tight on the right side [00:41:00] gently. I gonna lift the right side for a few second. Then going back, and this is what you know from the core on the other side. I’m gonna lift it up a little bit, few second and let it go.
We want to disengage the nozzle bone from the frontal bone. This is the first maneuver that you wanna do is the point where the fas of the brain is attached in the phy Ali,
and we just want do a distraction with a light force until I feel softening [00:42:00] from the two bones. This is what you can do in your clinic. Same. Same. We feel the movement and the tension on the zygomatic bones and the right side, and you just crawl and unscr those bones. Then the left one to find the movement.
We know the masse is attached there. Okay, so all the NJ dysfunction, you’re gonna block this part. We want to disengage that frontal zygomatic sutures and you’re gonna follow the rhythm of flexion and extension of the cranium and try to give some movement and remove the dural addition from those bones and sutures.
So where you feel restriction between two bones, you try. To let them together or [00:43:00] to move out then.
So really nice and easy. Maneuver. We got occiput. We got sphenoid. I gonna hold the occiput. I gonna hold the sphenoid. Hold the headaches. Three nerve problem to give a movement high here. Problem abalar synosis. I can hold the occiput and force inflection the foid.
Hold then do the opposite in extension. Then I can add rotation on one side, rotation on the other side, and then I can combine the occiput that’s gonna rotate on one side [00:44:00] and the sphenoid on the other side when they find a point of restriction. I can wait until the frequency is gonna do the work for me really easy.
In this case, you hold and you rotating and see what’s gonna happen. Oid, you can’t hurt HeartWise and the occiput anticlockwise. And wait and feel for the release. Then you learn from Dr. McMakin. I can ask for that Salva movement. So please tuck your S chin down gently. Inhale, hold the bread and push your head back.
This is to create space inside the cranium and to do a do release. I want do this to make space inside the cranium. Then you can mobilize [00:45:00] the neck now to move the neck and give some motion in the vertebra. I can do side bending. If there is no pain, I can do side bending. If there is no pain, I have some restriction in this side.
I can start to move
and then to move. And you can do a freestyle there, see where there is a restriction. You wait, you move, you turn side, bending, flexion, extension, and then you work on the temporalis, now the temporal bone and see if there are any restriction in there.
You can do a three fingers. You can maneuver in a easy way just [00:46:00] pulling the here and see if there is a restriction on one side or the other one. Remember here, it’s always on the right side. The tentorium is attaching there. You just pull the here and make some space and the hypothesis will say thanks to you.
So this is at a technique to work on the sphenoid, ethmoid and palate if you are allowed to do the intraoral maneuver. It is something that really works. If you’re not allowed, you can just work from outside of the cranium. You can work on the cranium really in a easy way, as I showed before, but if you are allowed to use your finger to do a intraoral maneuver, you can restart the cranio secular movement just.
Holding the sphenoid, go to the heart palate [00:47:00] and get in cotton contact with moid bone and I push down the sphenoid. I feel the moid that pushing on the heart palette, and then it can extend and push the heart palette. Push the ethmoid and push up this PHE night, I’m gonna follow the cranial rhythm. To do that really is a manual to do then we know that the doura is attached to the pelvis, the neck. It can be caused from a restriction in the pelvis, in the sacrum and the cox. So we are gonna now work on the hips to release the dura here on the pelvis. I’m gonna [00:48:00] gentle flex the knee 30 and 4 43 and you start to melt the hip. I feel tension at that point.
I can wait a little bit back. Remember the timing of frequency, so don’t be so fast. Give time to the frequency to do the job and then you just move when a little bit, the addition it’s and move. Out
and you find for a point of restriction, you do internal external rotation abduction, and see and check point for restriction to release a little bit on this side,
we’re gonna work on the other leg. [00:49:00] Of course you will find a difference for one hip or to the other one. Remember that on the first picture, the pelvis is twisted friction. I wait. I go back and nice and gently I move a little bit more. And this is the timing that you can reproduce during your treatment.
Asking also if the patient feel pain to the beauty of FSM, that everybody can obtain the same result, some rotation, using the same frequency, and push hold. Wait a little bit back. Wait. All right. We’re gonna work on, okay. So now let’s see what’s happened to the hip [00:50:00] rotation. We are checking now the result.
The internal rotation, you see the difference improved a lot. As you can see,
we check the other side, so not bad for 20 minutes of treatment improvement also. And we check the head rotation. We check the rotation of the head now one side, and you see how different is from before on the other side better. And we check for the hip flexion, see how smooth is the movement right now and the range of motion improved.
And on the other side as well, if I can have your attention for 10 more minutes. [00:51:00] I will show you the next step, what you have to do because it’s not finished. This is good result, but we remember that we have our spine. And so removing addition and sitting position is another chapter about the work. There is one towel under the feet, other tower on the neck, and we start, we wanna work for the dura Addition in sitting position.
We are setting with three machine, one for addition in the dura, one for addition in the cord, and one for sclerosis in adipose tissue because we know that there are some adipose tissue between those tissue and the spine. So with the neck in normal position, I put my hand on the DM and they’re gonna ask the patient, please bend slowly.
Until you [00:52:00] feel a tension and already here you see there is tension, how the back is straight. This part, like a straight line. Now we wait, we go back I
and gently we go a little bit further here. You have to be patient when there is a vision. You have to wait and let the frequency do the work. Wait and gentle.
We can go a little more
and back. You can add rotation and some rotation on this part really gently. With extension [00:53:00] and you can let the extension of the spine rotate and extend your spine and find the vertebrae and give some motion in there. And now flex a little bit down
and you can see that there is the scar of the implant that he had patient with that. So try to imagine what we have in that part of the body a little bit farther. 15 years of back pain. Release your hand.
We can release the hand and see if we can go a little bit. The next step is to release the hand of the patient and let the ax go a little bit farther. Only if he can, [00:54:00] now we can add a frequency to removing scar and arachnoid tissue and we can set the big machine for that. Okay. We can add a more, one more frequency for the arachnoid.
If you have multiple machine, you do. And this way, if you have only one machine, you do multiple treatment piano.
And now you see how the shape of the spine is start to change.
But still we have some tension back on the part of weight L four, L five, that’s one and down a little more.
Weight[00:55:00]
and little down the speed up. Breathe in and turn a little bit more. You can increase the rotation better. Yeah, lower it by lower. And that the patients start to understand, relax your hand the change and go down. Go. Then tell me if you can go down. Little more. Little more. So now you can see that the back start to get round, but still missing one little piece.
Wait second down there. And you got the feeling under your hand. Little more. We have a big restriction for this surgery and we are gonna try to do some side bending. You have to be creative to release the dura and some side bending [00:56:00] on that side. If he’s not moving one direction, try another direction. And again, side bending and rotation. And this is easy. Things made the change in him and back side bending and rotation
and back some extension. So I touch your diaphragm. Go back from there. Release your arm and gently if you can, go over down. And here is our result. Down. Down. And try to touch with your, numb, with your chest, [00:57:00] your leg. So this is what we want. Okay. Marble as a result. Thank.
Did it stay? Huh? Did it stay? I had no phone call. For the moment, yes, but of course is a treatment that you have to repeat. It’s not one visit fix. I was lucky in this because I learned really well from Carol. And then I’m gonna ask to Mattel now to do the movement for the last things. If the patient wants to come in your office, it can be by himself just with the machine, and he gonna do the movement by himself,
or your assistant can really show him how to move and he’s gonna do the movement by himself and you can increase your practice and the patient will be really happy to come. [00:58:00] Amazing. So back now and up we want to understand, but there are correlation between doura and posture or between the dura and the cranial nerves.
Is that real? So we have a big restriction. So this is the result. So see the convergence, that’s how different it is. And how on the post-treatment, the left eye is moving. So maybe we did something on the cranial nerves. And okay with wearing, we have a change in the computer analysis, but have a look in his posture.
The shoulders are level out, the pelvis is un twisted. The gravity line, it’s a little bit increase from behind. The two SI joints are on the same level, the two scapula are on the same level and see on the bending [00:59:00] test how it improves just in 30 minutes treatment that you can do in your office as well.
So I hope that even if I was the last one, one synapse. Start to happening in your brain, and I thank you so much for your attention.
Thank you. Any question? No. This time so he was, he had a spinal fusion. He was misdiagnosed as his so he had a spinal fusion that was misdiagnosed when it was really no, the neurosurgeon. They decide to fuse L five S [01:00:00] one because they think that disc was the cause of the problem. But after the surgery, the patient get sicker and sicker uhhuh, and they decide to remove something really strange, never happen.
Then they remove some implant in the spine, but they decide to remove and they do some thermal ablation about the nerve endings. But of course we know that the nerves will regrow in one way or another one, and the pain gonna start again. Wow. Okay. Thank you. Okay, my red light is on. Is this still working?
Okay. Okay. So if I could have Dr. David Snick come up to the stage, if I could have Mary Ellen Chalmers please. And did I see Kim? Did Kim step out?
Is Kim close? How about this? While we’re waiting for Kim to come in those of you who are in the room with us who are certified pr Kim, [01:01:00] come on up. Those of you who are certified practitioners, would you just stand up for a moment so the rest of the room can see who you are? Those of you who are certified in the room.
Okay. Wonderful. And stay standing if you would. Fabulous. Stay standing. And those of you who are also trained as instructors, your practicum instructors, could you also stand practicum trained instructors, please. Excellent. Great. And then stay standing for me and then anyone else in the room who will be presenting over the course of tomorrow with the advanced and then also into the weekend for the symposium, would you please stand?
Anyone here who’s gonna be on the stage yet over the next couple days? Great. So here’s the great news. Everyone, the group of you who are standing are the ones who I’m supposed to be reminding are coming to the six 30 reception. [01:02:00] Okay? So six 30 to eight, all of the certified, the instructors, and all the presenters.
This weekend you are being beckoned to the reception at six 30. Also for the rest of you we’ll be starting sharp tomorrow morning with our full day’s routine. But in thanking you all for being here, we’d like to acknowledge all four of our primary speakers today. And on behalf of Dr. McMahon Dear Kim, thank you so much.
Thank you. Wonderful morning. I know many of us wanted to be in two places at one time this morning. Mary Ellen, thank you. Thank you.
I don’t wanna drop one, so that’s why you see I’m not multitasking. Thank you all. Eduardo, that was brilliant. Thank you. And then Dr. Nik, thank you so much and he will be taking the stage again tomorrow morning. As we wrap up today, one thing I love to do with every group or any [01:03:00] class that I ever run is I wanna hear from you your aha moments.
So you had these like brilliant educators here, right? There had to be like these huge like explosion moments today. ’cause I was having my own, right? You had to have some big ahas, right? So who’s willing and bold enough to share their ahas for today? Who had an aha moment, like something that, like you connected the dots.
You learned something new today. Come on, be bold. Are you in,
that’s an amazing integration that you did. Ahas today, Dr. Chalmers presenting the concept or the noting that the dura, the tri nerve needs to be addressed. Bang. Wonderful, fabulous. Great. And what was your aha. Stand up if you don’t mind, so they can capture you. Sure. Oh, it was an easy one. The tapping app.
Ah, tap. Tap. I love that. That’s perfect. Great. [01:04:00] There have to be more. There’s 150 of you in here. Brilliant. Stand up if you don’t mind. What was your big aha? What did you connect or what blew you away today? The connection of the dura and a tailbone injury. I think everybody’s fallen on their tailbone.
I know I have. And I wonder if that’s contributing to my neck. Yeah, but everybody almost, when we were little roller skating? Yeah, roller skating. Oh, those were the good days, weren’t they? Yes. Ooh, the Izzy Dory roller rink in me. We had a relationship before kindergarten. Anybody else? Uhhas?
Yeah. Kim, in your presentation there, there were a lot of musculoskeletal frequencies that I did not think of using that I’m gonna try. Yay. Yeah. And then email me all the good results. Fabulous. So good to see you. The post COVID urinary frequency related to decreased a DH production. Oh, that’s a really big one.
Yeah. That is huge. And [01:05:00] that connection too. Back to mold. Great. Any other ahas you’d like to share with your peers? And thank you to everybody who’s been on camera all day with us today. Thank you. The trigeminal cave, the trigeminal case. But until you. I love it. The trigeminal cave. Fantastic. Makes us think about today is the beginning of the Eclipse corridor.
Those are my astrologists in the room. As I did a lot of anatomy. I never saw the holes from the top of the inside of the cranium. I fell on my back. Thank you. You’re welcome. Wonderful. Brilliant. Any other ahas to share with your peers and your speakers today? Fabulous. Eduardo, I just really love what you said about structure first, soft tissue after that.
And then figuring out what you’re gonna do in terms of the nutrition and everything else. To [01:06:00] support the whole thing. Yeah. So thank you for that. Oh, and don’t you think that Eduardo should create, like audio nighttime books so that we can listen to his voice and go to sleep? I want them. So all of you.
It was absolutely extraordinary today. Every single one of you, I was hyper impressed with the, all the images the last presentation, all the images. I had never seen that before. And the dura, the pia, the arachnoid, I had never actually seen it like that. And then the con connection to the occipital, I actually had a request today from a patient who just had neck surgery with, she’s my eyes are a mess.
So now I get to show her some of that imaging. So thank you. Wonderful. Yes.
I like to say everything was great and I think one of the things that really hit home with me, it was the sleep. Mainly because I myself have [01:07:00] sleep problems and I really appreciate hearing all about the red light, the blue light therapies, the winding down of the mind. And I’ve already got those things ordered
and I’ve already got something from Premier and I am going to test it out tonight. Yeah. Thank you very much. Thank you. Brilliant. Brilliant. So those of you who completed the core. And so this would be the second event in your journey into the tribe of microcurrent. Would you please stand so that the rest of us can welcome you home and into the tribe and talk to you over the rest of the weekend?
Make sure you know you have found your people.
Aw, welcome everyone. Wonderful. Thank you for an amazing day one. Dr. McMahon has assured us she will be ready to rock tomorrow morning. [01:08:00] Good to go. So those of you who have the evening free, get some fresh air, wiggle a little bit ’cause you know you got another big day sitting tomorrow. Have a wonderful night and we’ll see you all back tomorrow morning.
Thanks everyone. Thank you. Got a question? Yeah. So here’s my question. How important is it? Move over here. Yeah.